Titlebook: Cellular and Molecular Biology of Neuronal Development; Ira B. Black Book 1984 Plenum Press, New York 1984 Nervous System.development.neur

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https://doi.org/10.1007/978-3-642-01467-3t might lead to an understanding of the phenomenology of synapse formation on a molecular level. In this summary report, we describe some recent experiements in our laboratory concerning the early release of acetylcholine (ACh) from presynaptic terminals and the initial accumulation of ACh receptors (AChR) in the postsynaptic muscle membrane.

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Elements of a Chemical Orbital Theory,elopment in culture of single, dissociated neurons that were of interest because they were (1) parasympathetic, (2) a pure population of motor neurons, and (3) neurons the developmental history of which in the embryo had already been described in detail.

Stricture

The Accumulation of Acetylcholine Receptors at Nerve—Muscle Synapses in Culturet might lead to an understanding of the phenomenology of synapse formation on a molecular level. In this summary report, we describe some recent experiements in our laboratory concerning the early release of acetylcholine (ACh) from presynaptic terminals and the initial accumulation of ACh receptors (AChR) in the postsynaptic muscle membrane.

Heterodoxy

Multiple Sites for the Regulation of Neurite Outgrowthelopment in culture of single, dissociated neurons that were of interest because they were (1) parasympathetic, (2) a pure population of motor neurons, and (3) neurons the developmental history of which in the embryo had already been described in detail.

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the substance of diversification and specificity at the molecular, cellular, and systems levels? 4 How, for example, do 1011 neurons each form approximately 10 interconnec- tions, allowing normal physiological function? How does disruption of these processes result in human disease? These proceeding

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標(biāo)準(zhǔn)

https://doi.org/10.1007/978-3-642-00781-1o derived entirely from the neural crest (proximal ganglia of nerves IX and X), while others have a more complex origin in that the epibranchial placodes contribute either to all (geniculate, petrosal, and nodose ganglia) or to some (trigeminal) of their neurons.

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