Titlebook: Cellular and Molecular Biology of Myelination; Gunnar Jeserich,Hans H. Althaus,Thomas V. Waehneld Conference proceedings 1990 Springer-Ver

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https://doi.org/10.1007/978-3-642-00331-8Gra?a and Blakemore, 1986). This lesion therefore provides a system in which one can examine interactions between axons and glia which are involved in the reconstruction of a CNS environment around axons. By injecting glial cell cultures of differing composition it is possible to examine the ability

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https://doi.org/10.1007/978-3-642-00331-81961) and Bornstein et al (1962) a spontaneous remyelination process was described in numerous animal models of demyelination (reviews by Blakemore 1979–1981, Ludwin 1987). In Multiple Sclerosis (MS) a discrete remyelination was first described at the periphery of chronic lesions (review by Prineas

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Orale Antibiotika in Klinik und Praxisity (Kuo et al., 1980) or amount (Girard et al., 1986; Yoshida et al., 1988) and by an abundant occurrence of its substrate proteins (Wrenn et al., 1980). It is likely, therefore, that PKC plays a pivotal role in neuronal function and regulation. In this chapter, I summarized some of our work on imm

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Cellular and Molecular Biology of Myelination978-3-642-83968-9Series ISSN 1010-8793

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1010-8793 Overview: 978-3-642-83970-2978-3-642-83968-9Series ISSN 1010-8793

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Regulation of Oligodendrocyte Progenitor Development: Antibody-Perturbation Studiesnerve conduction allows for dramatic savings in energy consumption and space utilization. Disruption of myelinogenesis causes serious neurological deficits, many with chronic implications (Morell, 1984).