Titlebook: Cellular and Molecular Biology of Mammary Cancer; Daniel Medina,William Kidwell,Elizabeth Anderson Book 1987 Plenum Press, New York 1987 b

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Aboveboard

Stem Cells in Mammary Development and Cancere mammary parenchyma within the mammary fat pad (4). Extension of the mammary ductal tree and the generation of its branching pattern occurs in three main ways in varying proportions: first by the linear lengthening of existing ducts, secondly by dichotomous branching of the growing ductal tips, and

mendacity

Silent-Ischemia

Chromosomes in Breast Cancerinterest in cytogenetic analysis. Tumor-specific aberrations could be valuable in identifying tissue-specific developmental genes and in detecting sites within the genome that are critical for cell growth or control of cell division. Documentation of tumor-chromosome associations seems essential to

optic-nerve

Epithelium — Mesenchyme Interaction in the Fetal Mammary Glandsues are responsible for the diversification of cell types; later, interaction between already committed cell populations is required for the precise development of the complex structures of vertebrate organs (Kratochwil, 1983). These organs are typically formed by at least two tissues of different

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內(nèi)向者

ambivalence

RAFF

Interaction of Mammary Tumor Subpopulationsment and functioning of normal, as well as malignant, mammary tissues. This environment consists of other cells, of extracellular matrices, and of a wide variety of growth modulating factors. Looked upon from this perspective, tumor subpopulation interactions are but a special case of a general biol

Rankle

Rodent Models To Examine,, Hormonal Regulation of Mammary Gland Tumorigenesisonstrated that mammary tumors were more frequent in outbred multiparous mice than in nulliparous mice of the same strain (1). Three years later, Lathrop and Leob reported that ovariectomy of mice at an early age either completely inhibited or greatly delayed the appearance of mammary tumors (2). Sin