Titlebook: Cellular and Molecular Basis of Synaptic Transmission; Herbert Zimmermann Conference proceedings 1988 Springer-Verlag Berlin Heidelberg 19

Reverie

978-3-642-73174-7Springer-Verlag Berlin Heidelberg 1988

PLAYS

overhaul

ETHER

Excitatory Amino Acids; Physiology, Anatomy and Biochemistryocysteate. All 3 candidates are released from brain slices in a Ca. dependent manner. Surprisingly enough there is a good positive correlation between the release of glutamate, aspartate and homocysteate from different regions of the brain (Do et al, 1986), as if they were all released from almost the same structures in the different regions.

不朽中國

Heterologous Carriers Located on Rat CNS Nerve Endings and their Possible Role in the Modulation of m is now well accepted. The nerve terminals of the central nervous system are also endowed with membrane transporters. It has been suggested that the main function of reuptake processes should be to remove the transmitter released previously into the synaptic cleft and to terminate its action at the postsynaptic receptors.

Lobotomy

DIKE

Adrenergic and Cholinergic Vesicles: Are there Common Antigens and Common Properties?red ones (SDV). In this short review we discuss the possibility that this resemblance is not only expressed at the morphological level but also by the biochemical and functional properties of these vesicles.

Glucose

想象

Mechanisms of Failure of Synaptic Transmission During AnoxiaEvidence from intracellular recording and voltage-clamping in hippocampal slices indicates that brief anoxia (2–4 min) strongly depresses excitability and synaptic potentials by raising K conductance and inactivating Ca-currents. Both effects could be caused by a rise in intracellular free Ca., but other explanations are not excluded.

為敵