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Titlebook: Cellular and Molecular Aspects of Inflammation; George Poste,Stanley T. Crooke Book 1988 Plenum Press, New York 1988 Lipid.immune system.i

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書目名稱Cellular and Molecular Aspects of Inflammation
編輯George Poste,Stanley T. Crooke
視頻videohttp://file.papertrans.cn/224/223084/223084.mp4
叢書名稱New Horizons in Therapeutics
圖書封面Titlebook: Cellular and Molecular Aspects of Inflammation;  George Poste,Stanley T. Crooke Book 1988 Plenum Press, New York 1988 Lipid.immune system.i
描述The characterization of the cellular and molecular mechanisms that mediate inflammation provides a foundation that supports future studies that will de- fine mechanisms more intimately. It encourages substantial optimism about the opportunities to understand the inflammatory process and to use that information to develop novel therapeutic approaches. Recent progress has defined the cells that mediate the inflammatory response, many of the inter- cellular transmitters, the receptors, signal transduction processes and regula- tory mechanisms. Thus, we now have the opportunity to understand inflammation in pharmacologic terms and to attack the key molecular targets to develop new therapeutics. Among the cells involved in the inflammatory response are the lympho- cytes, neutrophils and endothelial cells. Maintenance of homeostasis, re- sponse to proinflammatory stimuli and pathophysiologic responses are products of complex interactions between these and other elements of the immune systems. Each of these cells displays a variety of receptors to define the stimuli to which they respond. The receptors displayed that the signal transduction processes and cellular responses are regulated g
出版日期Book 1988
關(guān)鍵詞Lipid; immune system; inflammation; molecular aspects; receptor
版次1
doihttps://doi.org/10.1007/978-1-4684-5487-1
isbn_softcover978-1-4684-5489-5
isbn_ebook978-1-4684-5487-1
copyrightPlenum Press, New York 1988
The information of publication is updating

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978-1-4684-5489-5Plenum Press, New York 1988
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Professionelle Kundenkommunikation,emagglutinin (PHA) or concanavilin A (Con A) in cultures of murine thymocytes (Gery .., 1972). Subsequent studies revealed that IL-1 was stimulatory for a broad range of T-cell-dependent . immune responses including antibody synthesis (Koopman ..,1978; Staruch and Wood, 1983), cytotoxic T cell activ
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Erfolgreiche E-Mail-Kommunikation,pathological alterations in the physiology of their mammalian hosts. These disruptions of normal homeostasis, left unresolved, can lead to the depletion of host energy stores advancing to wasting (cachexia), tissue damage, multiple organ system failure, shock, and death. Although such symptomatic pa
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