Titlebook: Cellular Senescence; Methods and Protocol Marco Demaria Book 2019 Springer Science+Business Media, LLC, part of Springer Nature 2019 senesc

轉(zhuǎn)變

書目名稱Cellular Senescence影響因子(影響力)




書目名稱Cellular Senescence影響因子(影響力)學(xué)科排名




書目名稱Cellular Senescence網(wǎng)絡(luò)公開度




書目名稱Cellular Senescence網(wǎng)絡(luò)公開度學(xué)科排名




書目名稱Cellular Senescence被引頻次




書目名稱Cellular Senescence被引頻次學(xué)科排名




書目名稱Cellular Senescence年度引用




書目名稱Cellular Senescence年度引用學(xué)科排名




書目名稱Cellular Senescence讀者反饋




書目名稱Cellular Senescence讀者反饋學(xué)科排名

ALB

Conjuction

Tim M. Curtis Ph.D.,Tom A. Gardiner Ph.D. cells (Dimri et al., Proc Natl Acad Sci U S A 92:9363–9367, 1995). These cells, characterized by a permanent cell-cycle arrest (Hayflick and Moorhead, Exp Cell Res 25:585–621, 1961) and the production of a distinct secretory phenotype of cytokines, chemokines, and proteases (Coppe et al., PLoS Biol

VOC

Low-Coherence Interference Microscopy The method has been developed by Cawthon in 2002 (Cawthon, Nucleic Acids Res 30:47e–47, 2002) and remains the most frequently used technique either in original or modified version. Telomere length is estimated by comparing the amount of telomere repeat amplification product (T) to a single copy gen

Audiometry

Low-Coherence Interference Microscopyted with age-related pathologies such as cancer progression. Numerous functions of senescent cells depend on their ability to secrete bioactive molecules, a characteristic termed the senescence-associated secretory phenotype (SASP). Although the SASP is generally described as proinflammatory, its tr

Nonflammable

Superresolution in Scanning Optical Systems. In oncogene-induced senescence, inflammasomes play a crucial role by regulating IL1R signaling and consequently modulating proliferation and the senescence-associated secretory phenotype (SASP). Inflammasome activation requires two steps: (a) priming of the inflammasome by activation of . expressi

Nonflammable

magnanimity

Superresolution in Scanning Optical Systems suppressor mechanism limiting cancer progression. Here we describe IMR90 ER:RAS, a widely used model to study OIS in cell culture. This model takes advantage of IMR90 human primary fibroblast infected with a 4-hydroxy-tamoxifen (4-OHT) inducible ER:RAS construct. RAS activation upon 4-OHT treatment

Collision

brassy