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Titlebook: Cellular Ca2+ Regulation; Douglas R. Pfeiffer,Jeanie B. McMillin,Steve Littl Book 1988 The Editor(s) (if applicable) and The Author(s), un

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書(shū)目名稱Cellular Ca2+ Regulation
編輯Douglas R. Pfeiffer,Jeanie B. McMillin,Steve Littl
視頻videohttp://file.papertrans.cn/224/223008/223008.mp4
叢書(shū)名稱Advances in Experimental Medicine and Biology
圖書(shū)封面Titlebook: Cellular Ca2+ Regulation;  Douglas R. Pfeiffer,Jeanie B. McMillin,Steve Littl Book 1988 The Editor(s) (if applicable) and The Author(s), un
描述This book arose from a meeting held at the University of Washington, Seattle, in July of 1986. The meeting was a satellite symposium of the XXXth International Congress of Physiological Sciences which occurred in Vancouver, canada, at that time. 2 Adjustments in the cytoplasmic Ca + concentration of cells occur in response to a variety of external signals. These fluctuations are a cen- tral component of one mechanism by which cells adapt their activities to changes in the external environment and to the requirements of whole body 2 homeostatic mechanisms. It is now clear that redistribution of Ca + within 2 intracellular compartments, as well as changes in the rates of Ca + influx and extrusion at the whole cell level, occur during signal-dependent 2 changes in the cytoplasmic Ca + concentration. In summarizing current research in this area, this volume considers first the properties of indi- vidual cation transporting activities located in various cell membranes. It then moves to the cellular level, where the consequence of individual transporting activities acting in concert is examined. l!D.phasis is also 2 p1 aced on pa tho1 ogica1 conditions which resu1 t in loss of cell Ca +
出版日期Book 1988
關(guān)鍵詞cell; cell membrane; cells; cytoplasm; development; distribution; environment; ion transport; iron; membrane;
版次1
doihttps://doi.org/10.1007/978-1-4757-0007-7
isbn_softcover978-1-4757-0009-1
isbn_ebook978-1-4757-0007-7Series ISSN 0065-2598 Series E-ISSN 2214-8019
issn_series 0065-2598
copyrightThe Editor(s) (if applicable) and The Author(s), under exclusive license to Springer Science+Busines
The information of publication is updating

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The Role of Calcium and Phospholipase A2 in Glucagon-Induced Enhancement of Mitochondrial Calcium Reserved during isolation of the mitochondria (see 1 for review). The mitochondrial actions of glucagon may not be directly mediated by a rise in the level of cAMP; instead, mitochondrial calcium uptake may be involved in transferring the hormonal signal to the mitochondrial matrix. Glucagon induces a
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Regulation of Liver Plasma Membrane Ca2+ Pumpchanisms responsible for extrusion of Ca. out of the cell against its electrochemical gradient (for a review, see ref. 1). The extracellular free Ca. concentration is about 10. times that of intracellular free Ca. (50–200 nM). Therefore, the maintenance of a low intracellular free Ca. level is criti
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