Titlebook: Cell death in biology and pathology; I. D. Bowen,R. A. Lockshin Book 1981 Chapman and Hall Ltd 1981 DNA.DNA repair.biology.cell.cell cycle

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Michel Jacob,Gabriel-Assad Singaby in abnormal circumstances and producing manifestations of disease (see reviews by Trump and Mergner, 1974; Jennings, Ganote and Reimer, 1975). Their experimental models included disruption of metabolic regulation by various injurious agents, and their conclusions were orientated towards expanding o

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https://doi.org/10.1057/9780230594241ources of energy, nutrition and information? Nevertheless, it is now clear that substantial cell death is a normal part of embryonic development (reviewed in Saunders, 1966; Saunders and Fallon, 1967), and its frequent incidence is charted by Glücksmann (1951). Glücksmann distinguishes three types o

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Stefan P?tzl,Thomas Kohr,Michael Zollenkopry fission but also the loss of postmitotic cells by selective deletion and general senescence. At maturity, the volume and shape of most tissues containing mitotic cells is mainly a balance between the rate at which cells are produced and the rate at which they die, death occurring either in the ti

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Jürgen R. Grote,Achim Lang,Volker Schneider. It is implicated extensively in the death of whole plants, e. g. monocarpic plants which reproduce once only and die, or polycarpic plants which reproduce either a few or many times before dying, and with the death of parts of polycarpic plants. This latter point is seen in the case of flower, lea

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,Religious Diplomacy and Socialism, 1956–9,imultaneous progress along three different lines of hormone research provided new approaches to this problem. The first was the discovery of the second messenger, identified as the cyclic nucleotides, endowed with the property of transducing different hormonal signals in intracellular biochemical ch

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