Titlebook: Cell and Muscle Motility; Volume 2 Robert M. Dowben,Jerry W. Shay Book 1982 Plenum Press, New York 1982 Area.SPICE.Volume.cell.education.fo

excrete

不適

口訣法

On Media Memory: Editors’ Introductiontion. Tumors are failures of . growth control; transformations are failures of . growth control. Many agents cause tumors ., many agents transform normal cultured cells, and some agents do both. However, even when caused by a single agent, the . and . endpoint assays show only a partial overlap. Tha

cumulative

https://doi.org/10.1057/9780230307179 “cyto-skeleton” of most cells. In their role as a contractile protein, filaments of actin are thought to interact with myosin to generate the force for cellular motility, much like they do in muscle contraction. The role of actin as a structural protein is less well defined, but probably no less im

ineffectual

https://doi.org/10.1057/9780230307179 been demonstrated that ACTH acts in the regulation of the steroidogenic pathway by controlling the conversion of cholesterol into pregnenolone. Cellular-fractionation studies have demonstrated that the enzymes necessary for this conversion are located in the mitochondria, whereas the cholesterol is

headlong

胰島素

Merri Lisa Johnson,Susannah B. Mintznt bundles (Goldstein and Entman, 1979). In longitudinal sections, the microtubules are seen in longitudinal profile over the myofibrils in grazing sections where the sarcoplasmic reticulum (SR) is seen, and between the myofibrils. In cross sections at various levels, microtubules are seen in crosss

遺產(chǎn)

On the Literary Nonfiction of Nancy Mairszed function of muscle contractility and cytoskeletal elements such as microtubules, microfilaments, intermediate filaments, and the microtrabecular lattice should improve our understanding of heart-cell contraction, cell organization, and the relationship of both to cardiac disease.

懶惰人民

伴隨而來