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Titlebook: Cell and Molecular Biology of Endometrial Carcinoma; Hiroyuki Kuramoto (Professor),Masato Nishida (Pres Book 2003 Springer Japan 2003 angi

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31#
發(fā)表于 2025-3-26 22:31:21 | 只看該作者
HEC-1 Cells: Establishment of an In Vitro Experimental System in Endometrial Carcinomaum and endometrial carcinoma at the level of a simplified cellular system; contributing to cell and molecular biological studies on endometrial carcinoma. Once a cell line is established, it provides a stable experimental system that facilitates and progresses the study of the tissues and/or neoplas
32#
發(fā)表于 2025-3-27 04:53:54 | 只看該作者
33#
發(fā)表于 2025-3-27 08:43:43 | 只看該作者
Endometrial Stress Neuropeptides: Paracrine Effects on Cell Proliferation and Apoptosissteroids. Indeed, the two major types of human endometrial cells, namely epithelial and stromal, produce, among other neuropeptides, the stress-associated peptides corticotropin-releasing hormone (CRH), endorphins, and dynorphins. Multiple lines of evidence suggest that the stress-associated neurope
34#
發(fā)表于 2025-3-27 12:56:16 | 只看該作者
35#
發(fā)表于 2025-3-27 14:41:22 | 只看該作者
Involvement of Cell Cycle Regulators in Steroid Hormone-Induced Growth of Endometrial Carcinomandular cells and estrogen receptor (ER)/pregesterone receptor (PR)-positive endometrial carcinoma Ishikawa cells. The results indicated that the estradiol (E2)-induced upregulation of cydin Dl may play a crucial role in the growth of normal endometrial glandular cells and is mediated by c-Jun via an
36#
發(fā)表于 2025-3-27 19:21:58 | 只看該作者
37#
發(fā)表于 2025-3-27 23:54:23 | 只看該作者
Estrogen Receptor Signaling and Crosstalk with the Ah Receptor in Endometrial Cancer Cellsirth, indicating that “l(fā)ifetime” exposure to estrogens increases the incidence of both tumors. In contrast, smoking protects against the development of endometrial cancer, whereas the role of smoking in breast cancer incidence is equivocal and may be dependent on the timing and duration of smoking.
38#
發(fā)表于 2025-3-28 03:16:31 | 只看該作者
39#
發(fā)表于 2025-3-28 07:23:20 | 只看該作者
Inhibition of AKT Oncogenic Pathway in Endometrial Cancer Cellsctivation of AKT in a panel of endometrial cancer cell lines and tumor samples. Two endometrial cancer cell lines, namely Ishikawa and RL-95 cells, and several tumor samples showed elevated levels of phosphorylated AKT. The expression of PTEN significantly suppressed the growth of Ishikawa cancer ce
40#
發(fā)表于 2025-3-28 14:12:36 | 只看該作者
A New Paradigm for Vascular Endothelial Cell Growth Factor Receptor Signaling in Adult Endotheliumrocess that is dependent on the growth of new blood vessels. Outside of the female reproductive tract, the majority of the adult vasculature is quiescent and angiogenesis occurs only as part of the body’s repair processes or in diseases including diabetic retinopathy, rheumatoid arthritis, atheroscl
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