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Titlebook: Cell Impairment in Aging and Development; Vincent J. Cristofalo,Emma Hole?ková Book 1975 The Editor(s) (if applicable) and The Author(s),

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書目名稱Cell Impairment in Aging and Development
編輯Vincent J. Cristofalo,Emma Hole?ková
視頻videohttp://file.papertrans.cn/223/222831/222831.mp4
叢書名稱Advances in Experimental Medicine and Biology
圖書封面Titlebook: Cell Impairment in Aging and Development;  Vincent J. Cristofalo,Emma Hole?ková Book 1975 The Editor(s) (if applicable) and The Author(s),
描述lar aging, to which this model contributes, has grown. Apart from reports on work in this almost "classical" diploid cell system, the symposium presents studies using different biological systems with results that have been rewarding as information is obtained on patterns of change that are common to more than one experimental system. Indeed, in recent years much more has been learned about the fate of all different types of intermitotic and postmitotic cells in situ. The symposium has also presented contributions dealing, not directly with aging but with early ontogeny; such information on early developmental changes should certainly shed light on some of the mechanisms involved in aging. We are cognizant of the fact that environmental influences resulting from the complexities of modern civilization may have results that only occur much later, and profoundly affect the lifespan of the organism. There remain, of course, many unanswered questions. Whether there is "physiological" as opposed to "pathological" aging; whether "old" cultures living in unchanged, although not exhausted, medium, are degenerating, not aging; what is involved when "old" fragment cultures regenerate after e
出版日期Book 1975
關(guān)鍵詞aging; attention; bone; cell; cells; development; fat; kidney; retina; tissue; tumor
版次1
doihttps://doi.org/10.1007/978-1-4757-0731-1
isbn_softcover978-1-4757-0733-5
isbn_ebook978-1-4757-0731-1Series ISSN 0065-2598 Series E-ISSN 2214-8019
issn_series 0065-2598
copyrightThe Editor(s) (if applicable) and The Author(s), under exclusive license to Springer Science+Busines
The information of publication is updating

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https://doi.org/10.1007/b100384mparatively more attention has been directed towards the limited division potential of human fibroblasts than to limits in specific functional capability of this or other cell types with time in culture (1). This may be understandable when one considers the dearth of information concerning factors,
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https://doi.org/10.1007/b100384 shown age-associated increases in glycogen content (1), lipid synthesis (2), lipid content (3), the number of lysosomes (4,5) and the specific activities of lysosomal enzymes (6–8). In addition, age related decreases in the specific activities of transketolase and 6-phosphogluconate dehydrogenase h
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G. Bosco,A. Carena,V. Curri,P. Poggioliniotype. Conversely, abnormal cells survive indefinately (1). Several biochemical parameters have been studied in the past 10 years though very few of those studied have dealt with cell-surface integrity (2). However it is feasible to conceive that modifications of cell surface components would impair
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