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Titlebook: Cell Cycle Control; Michele Pagano Book 1998 Springer-Verlag Berlin Heidelberg 1998 Krebsforschung.Zellzyklus.apoptosis.cancer.cancer rese

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樓主: Abridge
31#
發(fā)表于 2025-3-26 22:32:34 | 只看該作者
Risk due to Miscellaneous Hazards, understanding the function of E2F transcriptional activity in growth control. In vivo and in vitro, E2F transcriptional activity appears to be complex, often capable of completely opposing actions. The basis for positive and negative action at many levels appears to be the ability of E2F transcript
32#
發(fā)表于 2025-3-27 03:25:01 | 只看該作者
33#
發(fā)表于 2025-3-27 07:53:45 | 只看該作者
34#
發(fā)表于 2025-3-27 13:24:33 | 只看該作者
https://doi.org/10.1007/978-3-540-69686-5Krebsforschung; Zellzyklus; apoptosis; cancer; cancer research; cell; cell cycle; phosphorylation; protein; r
35#
發(fā)表于 2025-3-27 14:28:20 | 只看該作者
36#
發(fā)表于 2025-3-27 21:04:44 | 只看該作者
Regulation of G1 Phase,ease in mass), chromosomal duplication, chromosomal segregation, and cellular division (Mitchison 1971). The components comprising the cell cycle engine should be distinguished from the downstream events they control. The engine is responsible for periodic cycling and is composed of biochemical circ
37#
發(fā)表于 2025-3-28 00:14:16 | 只看該作者
38#
發(fā)表于 2025-3-28 06:00:17 | 只看該作者
39#
發(fā)表于 2025-3-28 07:46:51 | 只看該作者
Regulation of the cell cycle by CDK inhibitors,ey might determine the duration of G1 phase by setting a threshold above which G1 CDK activity must accumulate before initiation of S-phase. In this threshold role, steady-state levels of CDK inhibitors (CKIs) counteract the temporal accumulation of G1 cyclin/CDK complexes. Second, they might act as
40#
發(fā)表于 2025-3-28 12:45:29 | 只看該作者
Regulation of the Cell Cycle by the Ubiquitin Pathway,on of key substrates by the ubiquitin pathway controls cell cycle progression, circadian rhythms, cell fate commitment in development, signal transduction pathways, and immune responses (see Hochstrasser 1995 for review).
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