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Titlebook: Cell Cycle Control; Michele Pagano Book 1998 Springer-Verlag Berlin Heidelberg 1998 Krebsforschung.Zellzyklus.apoptosis.cancer.cancer rese

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書(shū)目名稱Cell Cycle Control
編輯Michele Pagano
視頻videohttp://file.papertrans.cn/223/222782/222782.mp4
概述Addresses an important and fast-moving field
叢書(shū)名稱Results and Problems in Cell Differentiation
圖書(shū)封面Titlebook: Cell Cycle Control;  Michele Pagano Book 1998 Springer-Verlag Berlin Heidelberg 1998 Krebsforschung.Zellzyklus.apoptosis.cancer.cancer rese
描述Leafing through a journal of biology will almost certainly turn up several arti- cles on some aspect of the eukaryotic cell division cycle. The progress in un- derstanding the modus operandi of the cell cycle machinery and its central role in controlling cell proliferation has awakened a great deal of interest in many biologists. However, the cell cycle field often seems unnecessarily complicated, perhaps because data are acquired in bits and pieces and almost all simultane- ously from studies using different systems and organisms. The aim of this book is to make the background of the cell cycle field as unambiguous and clear as possible by distinguishing what is commonly accepted in the field from what are the unsolved problems. Therefore, this book is intended to serve a large au- dience: graduate and postgraduate students, basic and clinical scientists in- volved in the study of cell growth, differentiation, senescence, apoptosis, and cancer. My hope is that this book will stimulate further interest in the fascinat- ing field of the cell cycle and help scientists find connections between this and their fields of interest. This book brings together experts to review all major asp
出版日期Book 1998
關(guān)鍵詞Krebsforschung; Zellzyklus; apoptosis; cancer; cancer research; cell; cell cycle; phosphorylation; protein; r
版次1
doihttps://doi.org/10.1007/978-3-540-69686-5
isbn_softcover978-3-662-21695-8
isbn_ebook978-3-540-69686-5Series ISSN 0080-1844 Series E-ISSN 1861-0412
issn_series 0080-1844
copyrightSpringer-Verlag Berlin Heidelberg 1998
The information of publication is updating

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Regulation of CDKs by phosphorylation,her mechanisms participate as well. In contrast, extrinsic signals impinge on the cell, resulting in stimulation or inhibition of the cell cycle. These signals typically affect the levels of CDK inhibitors.
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Regulation of the cell cycle by CDK inhibitors,age-specific functions. Fourth, they might function as a gatekeeper of quiescence, keeping the GO cell.from inappropriately resuming proliferation. In some cases, tumors arise from well differentiated and non-mitotic cells that inexplicably return to the cell cycle. Over the last 5 years, biochemica
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https://doi.org/10.1007/978-94-017-2466-1her mechanisms participate as well. In contrast, extrinsic signals impinge on the cell, resulting in stimulation or inhibition of the cell cycle. These signals typically affect the levels of CDK inhibitors.
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