Titlebook: Cell Calcium Metabolism; Physiology, Biochemi Gary Fiskum Book 1989 Plenum Press, New York 1989 ATP.Calcium.biochemistry.chemistry.clinical

ERUPT

Angioplasty

https://doi.org/10.1007/978-94-011-1912-2acteristic behavior under sodium dodecyl sulfate polyacrylamide gel electrophoresis (SDS-PAGE); its apparent molecular weight was higher (165 kDa) under nonreducing conditions than under disulfide-reducing conditions (135 kDa) that were in the presence of dithiothreitol or.-mercaptoethanol. The Lazd

Flawless

Dale D. Ellis,John R. Preston,Heinz G. Urbannnels in skeletal muscle and some neuronal preparations (Schmid.., 1985; Chad and Eckert, 1986). The regulation of Ca channels by mechanisms implicating protein kinases other than cAMP-dependent protein kinase also has been reported (Di Virgilio.., 1986; Paupardin-Tritsch.., 1986; Rane and Dunlap, 1

MEN

Dale D. Ellis,John R. Preston,Heinz G. Urbana pump ATPase of SR by augmenting the key elementary steps of ATPase (Tada.., 1979, 1980). Based on steady-state and presteady-state kinetic analysis of the Ca pump ATPase, phospholamban was proposed to function as a regulatory cofactor of the Ca pump ATPase type I, found in SR of cardiac and slow-c

lipoatrophy

Current Issues in Production Ecologyrticular emphasis on how they might interact with each other to produce a highly integrated signaling network. One consequence of such interactions is that the second messengers may oscillate at variable frequencies, which may be an important element in how they mediate their effects. Before conside

Carcinoma

https://doi.org/10.1007/978-94-011-2534-5mber of diverse cell types in which the same GTP-activated Ca. release process is observed. In each cell type, submicromolar GTP concentrations rapidly effect a substantial release of Ca. sequestered via internal Ca. -pumping activity within a nonmitochondrial organelle, believed to be the ER. The C

Compatriot

Book 1989mechanisms responsible for 2 2 regulating intracellular Ca + and the pharmacological modalities for controlling cell Ca + . Particular attention is given to the molecular basis for plasma membrane transport activities, 2 2 including the ATP-driven Ca + pump, the Na + -Ca + exchange system, and volta

LAPSE

transport mechanisms responsible for 2 2 regulating intracellular Ca + and the pharmacological modalities for controlling cell Ca + . Particular attention is given to the molecular basis for plasma membrane transport activities, 2 2 including the ATP-driven Ca + pump, the Na + -Ca + exchange system, and volta978-1-4684-5600-4978-1-4684-5598-4

Hyperplasia

The High-Affinity (Ca2+ —Mg2+ )ATPase of Rat Liver Plasma Membrane Hydrolyzes Extracellular ATP ATP hydrolysis involves an enzyme phosphate intermediate. As a result of such a property, both the Ca. -stimulated ATP hydrolysis and Ca. transport can be inhibited by vanadate, a transition state analog of phosphate.

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