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Titlebook: Cell Adhesion Molecules; Cellular Recognition Martin E. Hemler,Enrico Mihich Book 1993 Springer Science+Business Media New York 1993 Activa

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書目名稱Cell Adhesion Molecules
副標題Cellular Recognition
編輯Martin E. Hemler,Enrico Mihich
視頻videohttp://file.papertrans.cn/223/222729/222729.mp4
叢書名稱Pezcoller Foundation Symposia
圖書封面Titlebook: Cell Adhesion Molecules; Cellular Recognition Martin E. Hemler,Enrico Mihich Book 1993 Springer Science+Business Media New York 1993 Activa
描述The Fourth Annual Pezcoller Symposium entitled Adhesion Molecules: Cellular Recognition Mechanisms was held in Rovereto, Italy, June 24-26, 1992 and was focussed on the detailed mechanisms whereby cells utilize certain integral membrane proteins to perceive their surrounding environment and interact with it. With timely presentations and stimulating discussions this Symposium addressed the genetics and biochemistry of adhesion molecules, the regulation of their functions and their role in cancer and the immune system. Emphasis was given to adhesion proteins in the integrin family because of the widespread distribution of this group of molecules and its important role in essentially all eukaryotic biological systems. The regulation of integrin genes and their expression are discussed in detail, as are specific aspects of the genetics of fibronectin. The molecular basis for the regulation of certain integrins, the function of these proteins in determining cell adhesion, and the consequences of this adhesion for the function of the cells involved are discussed. The role of certain integrins in stimulating signal transduction, the essential involvement of integrins in conditioning the
出版日期Book 1993
關鍵詞Activation; Immune System; biochemistry; carcinoma; genes; genetics; proteins; proteoglycans; regulation; sig
版次1
doihttps://doi.org/10.1007/978-1-4615-2830-2
isbn_softcover978-1-4613-6224-1
isbn_ebook978-1-4615-2830-2
copyrightSpringer Science+Business Media New York 1993
The information of publication is updating

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G. Itelson – A Socratic Philosopherdent, specific and saturable platelet receptor (Mustard et al., 1978; Marguerie et al., 1979; Bennett and Vilaire, 1979), and the failure of thrombasthenic platelets to bind fibrinogen and aggregate upon activation (Mustard et al., 1979), suggested a role for GPIIb and/or GPIIIa as part of the plate
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G. Itelson – A Socratic Philosopherreduce tissue damage and alter survival (Ferguson et al., 1991; Rosen, 1989; Vedder et al., 1988; Vercellotti et al., 1983). Thus, understanding the mechanisms of leukocyte exit from the bloodstream and activation at inflammatory sites is presently a subject of intense effort in several laboratories
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Dmitri Fursaev,Dmitri Vassilevich (for instance by alteration of the organization of the adhesion receptors at the cell surface), directly affect cell adhesion. Second messengers play a pivotal role in integrin activation, although at present the precise intracellular circuits that regulate integrin mediated cell adhesion are not c
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,Regulation of β1 Integrin-Mediated Adhesive Functions,s and histogenesis (Darribière, T. et al., 1990), leukocyte activation (Davis, L.S. et al., 1990;), cytotoxic T lymphocyte activity (Clayberger, C. et al., 1987; 1990; Shimizu, Y. et al., 1989), and lympho-hemopoiesis (Miyake, K. et al., 1991).
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