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Titlebook: Cardiovascular Hemodynamics; An Introductory Guid Arman T. Askari,Adrian W. Messerli Book 2019Latest edition Springer Nature Switzerland AG

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發(fā)表于 2025-3-21 19:17:47 | 只看該作者 |倒序瀏覽 |閱讀模式
書目名稱Cardiovascular Hemodynamics
副標題An Introductory Guid
編輯Arman T. Askari,Adrian W. Messerli
視頻videohttp://file.papertrans.cn/222/221948/221948.mp4
概述Includes new coverage on noninvasive assessment and the effects of interventions on cardiovascular hemodynamics.Provides board-style questions, clinical cases, and vignettes.Discusses diagnosis, evalu
叢書名稱Contemporary Cardiology
圖書封面Titlebook: Cardiovascular Hemodynamics; An Introductory Guid Arman T. Askari,Adrian W. Messerli Book 2019Latest edition Springer Nature Switzerland AG
描述The second edition of this key resource provides a broad and fundamental overview of basic cardiovascular (CV) hemodynamic principles with a focus on clinical assessment of CV physiology. Extensively updated, the book includes new coverage on noninvasive hemodynamic assessment and the effects of selected interventions on CV hemodynamics. It provides an introduction to the basic concepts such as preload, afterload, myocardial contractility, and cardiac output. Subsequent chapters examine the effects of interventions such as vasodilators, beta blockers, pressor agents, inotropes, and different forms of invasive circulatory support. The book also focuses on various methods of hemodynamic evaluation including echocardiography, CT/MRI, noninvasive hemodynamic assessment, and cardiac catheterization. The book concludes with a discussion of proper diagnosis, evaluation, and management of patients using hemodynamic data on a variety of specific disease states..?.An invaluable contribution to the. Contemporary Cardiology .Series., .the .Second Edition. of.?Cardiovascular Hemodynamics: An Introductory Guide.?is an essential resource for physicians, residents, fellows, medical students, and r
出版日期Book 2019Latest edition
關(guān)鍵詞Assessment of cardiovascular hemodynamics; Cardiovascular interventions; Cardiovascular performance; Ca
版次2
doihttps://doi.org/10.1007/978-3-030-19131-3
isbn_softcover978-3-030-19133-7
isbn_ebook978-3-030-19131-3Series ISSN 2196-8969 Series E-ISSN 2196-8977
issn_series 2196-8969
copyrightSpringer Nature Switzerland AG 2019
The information of publication is updating

書目名稱Cardiovascular Hemodynamics影響因子(影響力)




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發(fā)表于 2025-3-21 22:05:32 | 只看該作者
Afterload expel blood into the vasculature and can be understood in the whole heart as the stress encountered by left ventricular myofibers as they contract against the end-diastolic volume. Wall tension can be at least in part described by LaPlace’s equation and aids in the explanation of why a dilated vent
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發(fā)表于 2025-3-22 01:31:30 | 只看該作者
Renin–Angiotensin–Aldosterone Axis Modulators and Other Vasodilators in the pathophysiology of cardiac diseases, especially hypertension and systolic heart failure. Aldosterone leads to local effects of vasoconstriction and sodium resorption, and potentiates inflammation and fibrosis in several organs, including the heart. Inhibitors of the RAAS, particularly angiot
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發(fā)表于 2025-3-22 04:35:51 | 只看該作者
Beta Blockers and Calcium Channel Blockersrgic receptors at the nerve endings of the sympathetic nervous system and therefore reduce systolic pressure, heart rate, and cardiac contractility and output. Calcium channel blockers block voltage-gated calcium channels and inhibit the influx of calcium ions into cardiac and smooth muscle cells. T
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Recently Approved Pharmacologic Agents to Improve Outcomes in Heart Failureormonal inhibition on a background of hemodynamic effects. The agents that have been shown to be associated with improved outcomes in this patient population are inhibitors of the renin-angiotensin-aldosterone (RAAS) system, mineralocorticoids, and beta-blockers. More recently, two agents were FDA-a
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發(fā)表于 2025-3-22 19:20:50 | 只看該作者
Mechanical Circulatory Supports a state in which cardiac dysfunction is either wholly or partially responsible for an inadequate level of cardiac output to support end-organ function. CS is an especially dangerous state, with in-hospital mortality as high as 27–51% (van Diepen et al., Circulation. 136:e232–68, 2017). Therefore,
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