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Titlebook: Cardiovascular Genetics for Clinicians; P. A. Doevendans,A. A. M. Wilde Book 2001 Springer Science+Business Media Dordrecht 2001 DNA.ather

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書目名稱Cardiovascular Genetics for Clinicians
編輯P. A. Doevendans,A. A. M. Wilde
視頻videohttp://file.papertrans.cn/222/221943/221943.mp4
叢書名稱Developments in Cardiovascular Medicine
圖書封面Titlebook: Cardiovascular Genetics for Clinicians;  P. A. Doevendans,A. A. M. Wilde Book 2001 Springer Science+Business Media Dordrecht 2001 DNA.ather
描述All physicians practicing medicine encounter patients suffering from cardiovascular disease. This book has been outlined in such a way that vascular surgeons, general internists, neurologists and cardiologists should be able to use it. The book covers the complete scope of cardiac diseases in addition to chapters on hypertension and atherosclerosis. In many patients there is a family history of cerebrovascular accidents, myocardial infarction or peripheral arterial disease. Also in patients reporting collaps, palpitations and arrhythmias the family is crucial and can provide clues to a genetic cause of the disease. This book is published to guide physicians in the process of determining whether a genetic component is likely to be present. Furthermore, information is provided what the possibilities and limitations of DNA diagnostic techniques are. Finally, the importance of newly identified categories of potential patients, i. e. gene carriers without symptoms or any inducible sign of disease, is highlighted. For some patients a genetic diagnosis is essential to determine appropriate therapy and for counseling? In some other diseases DNA diagnostic tools are available but the releva
出版日期Book 2001
關(guān)鍵詞DNA; atherosclerosis; cardiovascular; congenital heart disease; diagnostics; genetics; heart; lipoprotein; m
版次1
doihttps://doi.org/10.1007/978-94-010-1019-1
isbn_softcover978-94-010-3888-1
isbn_ebook978-94-010-1019-1Series ISSN 0166-9842
issn_series 0166-9842
copyrightSpringer Science+Business Media Dordrecht 2001
The information of publication is updating

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1-83 - 1-161: C2H5NO4.(1/2)DHO - C6H4BrD, understood. Many genes involved in lipoprotein metabolism have been identified, some of which are strongly related to atherosclerosis or cardiovascular disease. Nonetheless, atherosclerosis is a multifactorial disease with clear environmental determinants, but also with a strong and unavoidable gen
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1-252 - 1-303: DIO3 - Myoglobin,emia is a possible risk factor for arterial occlusive disease [3]. Most retrospective and prospective studies have demonstrated an association between mild hyperhomocysteinemia and arterial vascular disease [4]. Until now however it remains unclear whether mildly elevated homocysteine levels itself
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1.8 Glossary of general abbreviations,n occurs through the binding of the glycoprotein (Gp) Ib/IX/V platelet receptor to von Willebrand Factor (vWF) and/or direct binding of platelets by means of their collagen receptor (Gp Ia/IIa complex). These reactions determine a conformational change in the platelet Gp IIb/IIIa integrin, from a li
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