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Titlebook: Cardiovascular Disease, Volume 2; Molecular Medicine Qing K. Wang Book 2007 Humana Press 2007 Microarray.Proteomics.bioinformatics.cardiova

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發(fā)表于 2025-3-21 18:24:59 | 只看該作者 |倒序瀏覽 |閱讀模式
書目名稱Cardiovascular Disease, Volume 2
副標(biāo)題Molecular Medicine
編輯Qing K. Wang
視頻videohttp://file.papertrans.cn/222/221922/221922.mp4
概述Includes supplementary material:
叢書名稱Methods in Molecular Medicine
圖書封面Titlebook: Cardiovascular Disease, Volume 2; Molecular Medicine Qing K. Wang Book 2007 Humana Press 2007 Microarray.Proteomics.bioinformatics.cardiova
描述Cardiovascular disease is the leading cause of death in developed countries, but is quickly becoming an epidemic in such well-populated countries as China, India, and other developing nations. Cardiovascular research is the key to the prevention, diagnosis, and management of cardiovascular disease. Vigorous and cross-disciplinary approaches are required for successful card- vascular research. As the boundries between different scientific disciplines, particularly in the life sciences, are weakening and disappearing, a successful investigator needs to be competent in many different areas, including genetics, cell biology, biochemistry, physiology, and structural biology. The newly developed field of molecular medicine is a cross-disciplinary science that seeks to comprehend disease causes and mechanisms at the molecular level, and to apply this basic research to the prevention, diagnosis, and treatment of diseases and disorders. This volume in the Methods in Molecular Medicine series, C- diovascular Disease, provides comprehensive coverage of both basic and the most advanced approaches to the study and characterization of cardiovascular disease. These methods will advance knowledge
出版日期Book 2007
關(guān)鍵詞Microarray; Proteomics; bioinformatics; cardiovascular; gene transfer; genes; hypertension
版次1
doihttps://doi.org/10.1385/1597452130
isbn_softcover978-1-61737-829-4
isbn_ebook978-1-59745-213-7Series ISSN 1543-1894 Series E-ISSN 1940-6037
issn_series 1543-1894
copyrightHumana Press 2007
The information of publication is updating

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Optical Mapping of Shock-Induced Arrhythmogenesis in the Rabbit Heart With Healed Myocardial InfarcEW software, we mapped changes in transmembrane voltage during defibrillation shocks and identified the mechanisms of vulnerability and defibrillation in rabbit hearts with healed myocardial infarction (≥4 wk postinfarction).
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,Efficient Transfection of Primary Cells Relevant for Cardiovascular Research by nucleofection?,unique perspectives for novel experimental setups as therapeutic strategies. Herein we present protocols for the efficient nucleofection of human umbilical vein endothelial cells, human coronary artery endothelial cells, smooth muscle cells (e.g., pig vascular smooth muscle cells), neonatal rat card
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Apoptosis Assays,fragmentation of genomic DNA into nucleosomal fragments. Recognized by a variety of assays, activated caspases cleave many cellular proteins and the resulting fragments may serve as apoptosis markers. Finally, the mitochondria and the Bcl-2 family proteins play an important role in this process that
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