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Titlebook: Cardiovascular Disease; Cellular and Molecul Linda L. Gallo Book 1995 Springer Science+Business Media New York 1995 Lipid.Oxidation.cardiov

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Algebraic Model of Hadronic Structure,be mediated by binding of HDL to a specific cell-surface receptor. Through this receptor interaction, HDL may continually deplete peripheral cells of excess cholesterol, thereby preventing the buildup of cholesterol in artery wall cells that is an early feature of the developing atherosclerotic lesion.
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The Defect in HDL3 Mediated Efflux of Newly Synthesized Cholesterol is Associated with Impaired Actiisk and plasma levels of HDL-cholesterol.. In addition to unspecific cholesterol desorption from the plasma membrane., depletion of cholesterol may be achieved by binding of HDL apolipoproteins to specific surface receptors and subsequent induction of cholesterol transport from internal stores to the plasma membrane .
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Book 1995 countries to review and update research on cardiovascular disease. This group satisfied the symposium goals of formulating a more comprehensive and integrated picture of the events contributing to atherosclerosis and of exploring modified gene expression as an to understanding the causes of atheros
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