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Titlebook: Cardiovascular Calcification and Bone Mineralization; Elena Aikawa,Joshua D. Hutcheson Book 2020 Springer Nature Switzerland AG 2020 Aorti

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發(fā)表于 2025-3-21 18:46:16 | 只看該作者 |倒序瀏覽 |閱讀模式
書目名稱Cardiovascular Calcification and Bone Mineralization
編輯Elena Aikawa,Joshua D. Hutcheson
視頻videohttp://file.papertrans.cn/222/221908/221908.mp4
概述Discusses treatments in bone disorders and ongoing therapeutic efforts in cardiovascular calcification.Provides clinical strategies that may be applicable to many types of cardiovascular diseases.Exam
叢書名稱Contemporary Cardiology
圖書封面Titlebook: Cardiovascular Calcification and Bone Mineralization;  Elena Aikawa,Joshua D. Hutcheson Book 2020 Springer Nature Switzerland AG 2020 Aorti
描述The book covers the basic science and clinical aspects of cardiovascular calcification and bone mineralization. Cardiovascular calcification is the leading predictor of cardiovascular morbidity and mortality, with a predictive value more significant than blood lipid levels. The presence of calcific mineral in cardiovascular tissues alters biomechanical performance, increasing workload on the heart and potentiating atherosclerotic plaque rupture and subsequent heart attack and stroke. This book examines the role of calcification in cardiovascular disease covering topics such as calcification in the atherosclerotic plaques and aortic valves arteries and valves, aortic valve replacement, peripheral artery disease, imaging of early calcification and target discovery. In addition, various forms of ectopic calcification as well as mechanisms of bone mineralization are discussed..Cardiovascular Calcification and Bone Mineralization. is an essential resource for clinicians, researchers, and other medical professionals in cardiology, pathology, and biomedical engineering..
出版日期Book 2020
關鍵詞Aortic valve calcification; Atherosclerosis and calcification; Bone formation and calcification; Cardio
版次1
doihttps://doi.org/10.1007/978-3-030-46725-8
isbn_softcover978-3-030-46727-2
isbn_ebook978-3-030-46725-8Series ISSN 2196-8969 Series E-ISSN 2196-8977
issn_series 2196-8969
copyrightSpringer Nature Switzerland AG 2020
The information of publication is updating

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沙發(fā)
發(fā)表于 2025-3-21 23:29:09 | 只看該作者
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Differential Mechanisms of Arterial and Valvular Calcificationdeath worldwide. Arterial calcification impairs vascular resistance, thereby contributing to heart failure, and increases atherosclerotic plaque rupture risk contributing to heart attack and stroke. Calcification of the aortic valve leads to narrowing of the valve opening, reduced blood flow, and he
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發(fā)表于 2025-3-22 12:16:26 | 只看該作者
Calcifying Extracellular Vesicles: Biology, Characterization, and Mineral Formationmplicated in disease progression. EVs are secreted by various cell types and can be assayed from tissue, cell culture media, and biological fluids collected from animals and human subjects. A special subtype of EVs known as matrix vesicles has been studied as nucleating sites for mineralization in s
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發(fā)表于 2025-3-22 16:25:07 | 只看該作者
Role of Biomechanical Stress and Mechanosensitive miRNAs in Calcific Aortic Valve Diseasee, the current therapeutic options lack medical therapies and are limited to aortic valve (AV) replacement. AV calcification occurs preferentially in the fibrosa side of the valve, which is exposed to unstable hemodynamic conditions. In contrast, the ventricularis side is exposed to more stable hemo
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發(fā)表于 2025-3-22 21:33:33 | 只看該作者
The Role of Calcification in Peripheral Artery Diseaselity (Shu and Santulli, Atherosclerosis. 275:379–81, 2018; Norgren et al., J Vasc Surg. 45 Suppl S:S5–67, 2007). Arterial calcification has emerged as a key predictor of poor outcomes in patients with PAD. The medial calcification most commonly identified in lower extremity arteries represents a pat
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發(fā)表于 2025-3-23 04:57:08 | 只看該作者
Bioprosthetic Heart Valve Calcification: Clinicopathologic Correlations, Mechanisms, and Preventionalve. Their major limitation is structural degeneration caused by calcification. Calcification following implantation occurs in a tissue made vulnerable to calcification by the chemical treatment and physical changes induced during valve fabrication and their consequences following implantation. Dat
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