Titlebook: Cardiovascular Biology of Purines; Geoffrey Burnstock,James G. Dobson,Joel Linden Book 1998 Springer Science+Business Media New York 1998

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The G Protein-Coupled P2Y Receptors,ted the P2Y receptors and are distinguished individually by a numerical subscript. This family includes five phospholipase C-activating mammalian receptors, the P2Y., P2Y. P2Y., P2Y., and P2Y. receptors, that have been cloned and unambiguously shown to be activated by nucleotides after heterologous

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Valves

P2-Purinoceptors and Cardiac Functions,t, the role of adenosine and ATP in the control of cardiovascular function has been widely investigated. Although the determination of ATP actions in many tissues, including the myocardium, is complicated by its rapid catabolism to adenosine, it was already clear in early studies that ATP and adenos

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Neuromodulation in the Cardiovascular System,acetylcholine in sympathetic and parasympathetic nerves, ATP may be differentially released in response to nerve stimulation. ATP released from the nerve terminals of sympathetic neurones may contribute to neurogenic smooth muscle contraction through activation of P2X. receptors, or alternatively in

Deadpan

搖擺

P2 Purinoceptors and Regulation of the Function of Platelets, Erythrocytes and Mast Cells,or the development of new pharmacological agonists and antagonists (notably platelets), the molecular cloning of members of the P2Y family, the dissection of the signal transduction pathways (especially erythrocytes), and the identification recently of the elusive permeabilizing ATP receptor (mast c

Alveoli

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Recent Advances in Cardiac Adenosine Metabolism, cardiac adenosine formation is the intracellular dephosphorylation of AMR More than 90% of the adenosine formed is rephosphorylated to AMP via adenosine kinase (1.95 nmol/min/g) and only a small fraction (0.06 nmol/min/g) escapes the metabolic cycle between AMP and adenosine and is released into th

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