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Titlebook: Cardiomyopathies; Realisations and Exp J. F. Goodwin,E. G. J. Olsen (Consultant Histopath Book 1993 Springer-Verlag 1993 ADP-ATP-carrier.au

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發(fā)表于 2025-3-21 16:22:25 | 只看該作者 |倒序瀏覽 |閱讀模式
書目名稱Cardiomyopathies
副標(biāo)題Realisations and Exp
編輯J. F. Goodwin,E. G. J. Olsen (Consultant Histopath
視頻videohttp://file.papertrans.cn/222/221879/221879.mp4
圖書封面Titlebook: Cardiomyopathies; Realisations and Exp J. F. Goodwin,E. G. J. Olsen (Consultant Histopath Book 1993 Springer-Verlag 1993 ADP-ATP-carrier.au
描述This book reviews the basic knowledge about the cardiomyopathies and re-enforces the well-known definitions and classification of cardiomyopathies and specific heart muscle diseases, respectively. It emphasizes the importance of maintaining the classification into hypertrophic, dilated and restrictive cardiomyopathies, even though there are exceptions. The term "realisations" in the title is used in the sense of "achievements" and alludes to the progress in the understanding of heart muscle diseases over the last three decades and also to the areas of knowledge still not fully explored, for example, the as yet un- classified putative cardiomyopathies such as arrhythmogenic right ventricular dysplasia, long QT syndrome and syndrome X. The accurate and sensitive identification of major risk factors for sudden death in hypertrophic cardiomyopathy needs further study, as do the implications of a familial basis in some patients with dilated cardio- myopathy. The "expectations" mentioned in the title centre around the molecular biological aspects of viral myocarditis and dilated cardio- myopathy and their relationship to each other; around the auto- immune basis for dilated cardiomyopath
出版日期Book 1993
關(guān)鍵詞ADP-ATP-carrier; autoantibodies; cardiomyopathy; clinical research; diagnosis; gene; genetics; heart; heart
版次1
doihttps://doi.org/10.1007/978-3-642-77598-7
isbn_softcover978-3-642-77600-7
isbn_ebook978-3-642-77598-7
copyrightSpringer-Verlag 1993
The information of publication is updating

書目名稱Cardiomyopathies影響因子(影響力)




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Ljudmila A. Bordag,Ivan P. Yamshchikovmyocardial disorder with ventricular dilatation and reduced systolic function. In early studies [1] the diagnosis was not appreciated until congestive signs had appeared, but now, because of improved knowledge, better technology and diagnostic advances, diagnosis can be made at an earlier stage. The
板凳
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Miglena N. Koleva,Radoslav L. Valkovthe term “cardiomyopathy”, the investigation of the patient has become increasingly sophisticated. The initial classification was based on the ability of the clinician to define the anatomical structure and function following routine cardiac catheterisation including coronary arteriography. This lev
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Millimeter Wave High Gain Antennas,istic distribution of hypertrophy to the septum (asymmetric septal hypertrophy, ASH), abnormal systolic motion of the mitral valve apparatus (SAM) and particular myocardial features which include myofibrillar disarray [2]. It most frequently occurs in isolation, but it may also be associated with ma
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Richardson: How to Read Romance,le. The first systematic description of the condition was made by the pathologist Donald Teare in 1958 [1], from the post-mortem appearances of the hearts of 9 adolescents and young adults, 8 of whom had died suddenly. Teare drew attention to the disproportionate thickness of the interventricular se
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Temporal Frame Interpolation (TFI),d in the category of restrictive cardiomyopathy. It has been proposed that these conditions should be referred to as eosinophilic endomyocardial disease. Obliteration of the cavity is usually observed in advanced cases [1]. Excluded from the Task Force recommendation is amyloid disease; although pre
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Klaus-Peter Dinse,Tatsuhisa Katorly diastole for a given increase in volume. Pathological changes in endomyocardial disease (with or without hypereosinophilia) affect ventricular compliance by unknown mechanisms leading to a restrictive pattern of filling. This haemodynamic profile is usually documented by cardiac catheterisation
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https://doi.org/10.1007/978-3-642-77598-7ADP-ATP-carrier; autoantibodies; cardiomyopathy; clinical research; diagnosis; gene; genetics; heart; heart
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