Titlebook: Cardiac-Vascular Remodeling and Functional Interaction; Yukio Maruyama (Professor and Chairman),Masatsugu Book 1997 Springer Japan 1997 a

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Nurturing Science-based Venturesof the LV-DPVR appear to be structural, because the relation is generally unaltered by acute pharmacological or physiological manipulations. External influences on the LV-DPVR are primarily direct ventricular interaction and constraining forces from the pericardium. These external loads are more eas

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Nurturing Science-based Venturesteracts with neurohumoral-mediated control. EE cells respond to physical and humoral stimuli with release of several substances that have important inotropic actions on the myocardium. Specific electrophysiological properties determine the intracellular Ca. increase in response to external stimuli a

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https://doi.org/10.1007/978-1-84628-874-6 that both diastolic pressure and systolic pressure increase. However, a change in compliance alone can only result in a decrease of diastolic blood pressure. Several methods are available to derive total arterial compliance from aortic pressure and flow. The pulse pressure method, based on the two-

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Nurturing Science-based Ventures (0.69 ± 0.26 [SDI]. .. was 0.93 ± 0.08, suggesting that the external work of the left ventricle was nearly maximum at this condition. Similarly, .. was 0.98 ± 0.01, indicating that myocardial oxygen consumption was almost minimum. When dogs ran on a treadmill, their heart rate and cardiac output do

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Vascular Remodeling During Heart Failureof heart failure patients such as exercise intolerance and peripheral muscle fatigue. Potential mechanisms of peripheral vascular alterations are desensitization to neurohormonal activation and vascular remodeling.

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Myocardial Interstitial Collagen Matrix Remodeling in Response to a Chronic Elevation in Ventricular, both the myocardium and the ventricle become stiffer. Systolic function is preserved by the accompanying hypertrophy and enhanced contractility unless the increase in myocardial collagen consists of a significant degree of endomyocardial reparative fibrosis. In contrast, chronic volume overload in

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