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Titlebook: Cardiac Remodeling and Failure; Pawan K. Singal (Professor),Ian M. C. Dixon (Assoc Book 2003 Springer Science+Business Media New York 2003

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書目名稱Cardiac Remodeling and Failure
編輯Pawan K. Singal (Professor),Ian M. C. Dixon (Assoc
視頻videohttp://file.papertrans.cn/222/221822/221822.mp4
叢書名稱Progress in Experimental Cardiology
圖書封面Titlebook: Cardiac Remodeling and Failure;  Pawan K. Singal (Professor),Ian M. C. Dixon (Assoc Book 2003 Springer Science+Business Media New York 2003
描述According to the World Health Report (2000 http:/ /www. who. int/whr), of the 55 million deaths worldwide in 1999, more than 16 million were secondary to car- diovascular complications. With the prospect of world population increasing from the current level of 6 billion to 9 billion by the middle of this century, the burden of cardiac disease is going to increase astronomically. Furthermore, scientists are being challenged not only to reduce mortality, but also to improve quality of life. Thus, more than ever, intellectuals from different disciplines including biology, sociology, informatics and health care have to join forces to meet the mandate. The World Heart Congress with a focus on "Frontiers in Cardiovascular Health" held in Winnipeg during July 6-11, 2001, made a unique attempt to bring these specialists together to brainstorm and map out the course of action for cardiovascular research and health in the next century. Anytime there is a relative increase in the workload on the heart, there are adap- tive myocardial as well as humoral responses. When these adaptations or remodel- ing at the organ, subcellular or gene level, become inadequate for a proper tissue perfusion, th
出版日期Book 2003
關(guān)鍵詞Angiotensin II; atherosclerosis; atrial fibrillation; cardiovascular; cardiovascular function; gene thera
版次1
doihttps://doi.org/10.1007/978-1-4419-9262-8
isbn_softcover978-1-4613-4864-1
isbn_ebook978-1-4419-9262-8Series ISSN 1389-1774
issn_series 1389-1774
copyrightSpringer Science+Business Media New York 2003
The information of publication is updating

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978-1-4613-4864-1Springer Science+Business Media New York 2003
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Theaterarbeit in der Gesundheitsf?rderungin chronic volume overload heart failure might be due to an increased level of OFRs and/or decrease in the antioxidant status. We, therefore, studied the effects of chronic volume overload in the absence and presence of vitamin E (antioxidant) on cardiac function and contractility, cardiac malondial
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Theaterarbeit in der Gesundheitsf?rderunged to the SHR (spontaneously hypertensive rat), but unlike the SHR, all animals, male and female, develop fatal congestive heart failure, most in the second year of life. SHHF rats are now being widely used by the pharmaceutical industry to evaluate potential therapeutic agents for preventing the ma
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