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Titlebook: Cardiac Left Ventricular Hypertrophy; H. E. D. J. Keurs,J. J. Schipperheyn Book 1983 Martinus Nijhoff Publishers, Boston 1983 arrhythmia.a

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發(fā)表于 2025-3-21 19:56:51 | 只看該作者 |倒序?yàn)g覽 |閱讀模式
書目名稱Cardiac Left Ventricular Hypertrophy
編輯H. E. D. J. Keurs,J. J. Schipperheyn
視頻videohttp://file.papertrans.cn/222/221785/221785.mp4
叢書名稱Developments in Cardiovascular Medicine
圖書封面Titlebook: Cardiac Left Ventricular Hypertrophy;  H. E. D. J. Keurs,J. J. Schipperheyn Book 1983 Martinus Nijhoff Publishers, Boston 1983 arrhythmia.a
描述Left ventricular hypertrophy (LVH) is usually considered to be a compen- satory adjustment of heart muscle to an inreased work load. LVH develops in the course of valvular or congenital heart disease, or when part of the myocardium is damaged by long-standing ischemia or infarction. In the hypertrophied heart the muscle fibers increase in size, not in number. The fibers are found to contain a larger number of myofibrils and the cell organelles are larger. From epidemiologic studies it is known that even mild LVH is associated with myocardial ischemia, ventricular arrhythmias, and sudden cardiac death. Most cases of LVH show focal degenerative tissue changes including cellular atrophy, myofibrillar disorganization, interstitial fibrosis, and loss of intracellular connections. Myocardial dysfunction develops and, unlike the functional adaptive changes found in pure hypertrophy, is not reversible by surgical treatment of the valvular heart disease or medical correction of hypertension. Interstitial fibrosis, intracellular changes of musc Ie cells, and loss of contract ile tissue lead to poor mechanical function and undoubtedly increase the risk of ischemia, arrhythmias, or sudden deat
出版日期Book 1983
關(guān)鍵詞arrhythmia; artery; circulation; congenital heart disease; echocardiography; heart; heart disease; hyperten
版次1
doihttps://doi.org/10.1007/978-94-009-6759-5
isbn_softcover978-94-009-6761-8
isbn_ebook978-94-009-6759-5Series ISSN 0166-9842
issn_series 0166-9842
copyrightMartinus Nijhoff Publishers, Boston 1983
The information of publication is updating

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沙發(fā)
發(fā)表于 2025-3-21 23:52:48 | 只看該作者
板凳
發(fā)表于 2025-3-22 01:05:22 | 只看該作者
A Nonstandard Approach to Hydromechanics,esis..Volume overload in pregnancy, anemia, renal failure and, most commonly, obesity is an additional hypertrophogenic factor increasing wall stress by increasing LV chamber volume. The combination of hypertension and obesity is particularly unfavorable for the heart..Aging in itself appears to be
地板
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Mathematics and Its Applications still have some: thus any decrease in pressure below to critical pressure decreases subendocardial flow causing ischemia..In acquired hypertrophy the total cross-sectional area of the vascular bed is not increased. Myocardial flow per gram of muscle is usually normal; total flow to the hypertrophie
9#
發(fā)表于 2025-3-23 02:40:54 | 只看該作者
https://doi.org/10.1007/978-94-011-4168-0ted, suggesting some capillary growth. Measurement of coronary pressure and flow in dogs with experimental LVH also demonstrated a reduction of specific vascular resistance as predicted by Wearn. We measured specific vascular resistance in isolated beating rat hearts, taken from normal and spontaneo
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發(fā)表于 2025-3-23 08:12:15 | 只看該作者
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