Titlebook: Cardiac Adaptations; Molecular Mechanisms Bohuslav Ostadal,Naranjan S. Dhalla Book 2013 Springer Science+Business Media New York 2013 bioch

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書目名稱Cardiac Adaptations影響因子(影響力)




書目名稱Cardiac Adaptations影響因子(影響力)學(xué)科排名




書目名稱Cardiac Adaptations網(wǎng)絡(luò)公開度




書目名稱Cardiac Adaptations網(wǎng)絡(luò)公開度學(xué)科排名




書目名稱Cardiac Adaptations被引頻次




書目名稱Cardiac Adaptations被引頻次學(xué)科排名




書目名稱Cardiac Adaptations年度引用




書目名稱Cardiac Adaptations年度引用學(xué)科排名




書目名稱Cardiac Adaptations讀者反饋




書目名稱Cardiac Adaptations讀者反饋學(xué)科排名

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Molecular Mechanisms of Cardiac Developmente in the body as it is derived from four distinct pools of progenitors: the first heart field (cardiac crescent), the second heart field (SHF), the proepicardial organ and the cardiac neural crest. These progenitors differentiate into the different cell types that comprise the adult heart: cardiomyo

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Ontogenetic Aspects of Cardiac Adaptation to Chronic Hypoxiastion of the presumed cardiac impact will be, therefore, of considerable importance. Experimental results have clearly shown that the immature heart is significantly more tolerant to acute oxygen deficiency than the adult myocardium. However, the mechanisms of this difference have not yet been satis

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incredulity

Differences in Concentric Cardiac Hypertrophy and Eccentric Hypertrophyeactive mechanism to compensate for volume overload or pressure overload. An increase in pressure, common in hypertension or resistance training, results in a concentric hypertrophic phenotype whereas an increase in volume, as seen with valvular defects or endurance training, results in an eccentric

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Cardiac Adaptation to Volume Overloadhological conditions like atrio-ventricular valve insufficiency or systemic arterio-venous shunt. Experimental models of pure volume overload due to chronic mitral regurgitation in dogs or aorto-caval fistula in rodents show that this type of remodelling differs in many ways from pressure overload.

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