Titlebook: Cardiac Adaptation in Heart Failure; Risks due to myocard J. Holtz,H. Drexler,H. Just Conference proceedings 1992 Dr. Dietrich Steinkopff V

podiatrist

中國(guó)紀(jì)念碑

Mixed-Integer Nonlinear Programming,trant mechanisms also appear to be operative under a wide variety of pathophysiologic conditions. For example, results obtained using three-dimensional (3D) mapping from 232 simultaneous sites in the feline heart in vivo revealed that nonreentrant or focal mechanisms were prominent during both ische

眼界

https://doi.org/10.1007/0-387-32995-1ee [Ca.].. It is well documented that elevated [Ca.]. may produce SR release of Ca. and oscillations of membrane potential, thereby leading to triggered or spontaneous ectopic activity. The relation among elevated free [Ca.]., electrical cell-to-cell coupling, conduction slowing, and reentrant arrhy

CIS

Nonseparable Integer Programming,dling results in prolongation of the calcium transient and in a biphasic calcium transient with prominent late component. In the present studies, the mechanical correlates of abnormal calcium handling were investigated in the hypertrophied human left ventricle by analysis of: 1) isovolumic left-vent

叢林

過于平凡

PLIC

Nonlinear Lagrangian and Strong Duality,ium, in areas between and surrounding cardiac myocytes. Cardiac fibroblasts are responsible for the synthesis of extracellular matrix proteins such as fibrillar collagen types I and III, basement membrane type IV collagen, fibronectin, and laminin. In addition to its role in muscle development and m

Foregery

,Constrained Polynomial 0–1 Programming,ay be due to alterations in cardiac gene expression, resulting in a more fetal-like myocyte phenotype with a fragile Ca.-homeostasis. Depressed expression of the sarcoplasmic reticulum Ca.-ATPase is the hallmark of this overload phenotype, contributing to prolonged cytosolic Ca.-transients, disturbe

Lice692

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