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Titlebook: Cannabinoids and the Brain; Attila K?falvi (Faculty of Medicine) Book 2008 Springer-Verlag US 2008 Cannabinoid.Neuroscience.cannabinoids.e

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51#
發(fā)表于 2025-3-30 11:56:50 | 只看該作者
Ernest C. Peguita,Alexander M. Solntsevsence and highly compartmentalized cellular distribution of CB. receptors in neurons localized to corticolimbic areas, basal ganglia, cerebellum, and brainstem accounts for the majority of behavioral actions associated with cannabinoid drugs. The discovery of endocannabinoids led to an avalanche of
52#
發(fā)表于 2025-3-30 14:17:04 | 只看該作者
Smart Innovation, Systems and Technologiesabinoids, such as Δ.-THC, produce psychoactive effects through activation of neuronal CB. receptors, while CB. receptors mediate the immune properties of this class of drugs. The molecular, signaling, and trafficking properties of CB. receptors will be the focus of this review. The cloning of CB. re
53#
發(fā)表于 2025-3-30 17:26:21 | 只看該作者
54#
發(fā)表于 2025-3-31 00:38:47 | 只看該作者
55#
發(fā)表于 2025-3-31 01:06:29 | 只看該作者
Michael I. Zabezhailo,Yu. Yu. Truninnamely, anandamide and 2-arachidonoylglycerol) and their receptors (CB., CB. and TRPV. receptors), which altogether can be termed as “canonical knowledge”. Still, experimental findings often display mismatches with this canonical knowledge: in the last decade, a rapidly increasing number of studies
56#
發(fā)表于 2025-3-31 09:07:06 | 只看該作者
57#
發(fā)表于 2025-3-31 10:34:00 | 只看該作者
58#
發(fā)表于 2025-3-31 14:36:19 | 只看該作者
Marcelo Corrales,Mark Fenwick,Nikolaus Forgóctions are pivotal to fundamental developmental processes, including progenitor proliferation and fate specification, lineage segregation, neuronal migration, differentiation and survival, in the embryonic brain has just begun to emerge. Understanding the basic developmental and signaling principles
59#
發(fā)表于 2025-3-31 20:38:22 | 只看該作者
60#
發(fā)表于 2025-3-31 22:25:16 | 只看該作者
Marcelo Corrales,Mark Fenwick,Nikolaus Forgó lead to activation of the apoptotic cascade and subsequent neuronal demise. Cannabinoids have been demonstrated to confer neuroprotection both in vitro and in a number of in vivo paradigms of neurodegeneration including cerebral ischemia, hypoxia, seizures and experimental autoimmune encephalitis.
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