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Titlebook: Cancer and Inflammation; Douglas W. Morgan,Ulf J. Forssmann,Marian T. Nakad Book 2004 Birkh?user Verlag 2004 Chemokine.angiogenesis.cancer

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發(fā)表于 2025-3-21 16:37:39 | 只看該作者 |倒序瀏覽 |閱讀模式
書目名稱Cancer and Inflammation
編輯Douglas W. Morgan,Ulf J. Forssmann,Marian T. Nakad
視頻videohttp://file.papertrans.cn/222/221232/221232.mp4
概述Excellent overview of the interrelations between cancer and inflammation.International authorship
叢書名稱Progress in Inflammation Research
圖書封面Titlebook: Cancer and Inflammation;  Douglas W. Morgan,Ulf J. Forssmann,Marian T. Nakad Book 2004 Birkh?user Verlag 2004 Chemokine.angiogenesis.cancer
描述.How are cancer and inflammation interrelated mechanistically and clinically? Though extensive literature exists on the topic "Cancer and Inflammation", there are relatively few texts that have truly integrated the two in spite of the many common mechanisms shared by their processes. Certainly, areas such as cytokines, growth factors, proliferation, signal transduction and angiogenesis, for example, are found in both. Yet, the dynamics of how these common mechanisms are maybe interrelated in the pathologies of the two is not widely covered. Such coverage, as presented in this volume, may help further understanding and bring new approaches to therapeutics...The first section of the book discusses inflammatory mechanisms, studied in cellular and animal studies. The second part concentrates on clinical studies with antiinflammatory drugs in cancer treatment. The volume is written for biomedical researchers in the health care industry and in academia who are working in these areas..
出版日期Book 2004
關鍵詞Chemokine; angiogenesis; cancer; cancer treatment; cytokine; cytokines; growth; immunotherapy; inflammation;
版次1
doihttps://doi.org/10.1007/978-3-0348-7861-6
isbn_softcover978-3-0348-9600-9
isbn_ebook978-3-0348-7861-6Series ISSN 1422-7746 Series E-ISSN 2296-4525
issn_series 1422-7746
copyrightBirkh?user Verlag 2004
The information of publication is updating

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Inflammatory chemokines: their role in tumor growth and progression,ns (mostly 8-14 kDa) and can be classified by structure into four groups (C, CC, CXC, CX.C) defined by the number and spacing of cysteines in highly conserved positions in the N-terminal region of the protein.
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Cyclooxygenase activity in gastrointestinal cancer development and progression: prospects as a therers of the gastrointestinal tract were responsible for more than 40% of all cancer deaths, nearly 2.2 million people [., .]. Interestingly, the incidence of these cancers can vary considerably between regions. This is most likely due to a variety of factors including environmental exposures, diet, s
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Inflammation, necrosis, and cancer, but spontaneous. Cancer is generally thought of as disordered cell growth driven by the accumulation of genes that confer a growth advantage or prevent cellular senescence [.]. Inflammation is thought of primarily as a non-immune, acute host-defense process. When persistent, chronic inflammation an
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1422-7746 ed mechanistically and clinically? Though extensive literature exists on the topic "Cancer and Inflammation", there are relatively few texts that have truly integrated the two in spite of the many common mechanisms shared by their processes. Certainly, areas such as cytokines, growth factors, prolif
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