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Titlebook: Cancer a Comprehensive Treatise 2; Etiology: Viral Carc Frederick F. Becker Book 1975 Springer Science+Business Media New York 1975 DNA.car

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發(fā)表于 2025-3-21 18:55:02 | 只看該作者 |倒序?yàn)g覽 |閱讀模式
書(shū)目名稱Cancer a Comprehensive Treatise 2
副標(biāo)題Etiology: Viral Carc
編輯Frederick F. Becker
視頻videohttp://file.papertrans.cn/222/221221/221221.mp4
叢書(shū)名稱Cancer, a compresensive treatise
圖書(shū)封面Titlebook: Cancer a Comprehensive Treatise 2; Etiology: Viral Carc Frederick F. Becker Book 1975 Springer Science+Business Media New York 1975 DNA.car
描述The impact of basic research on oncology has been particularly impressive in the recent search for the cause of malignancy. Equally impressive is our appreciation of the cause of tumors based on observation. Even in the earliest era of the study of infectious diseases, it was proposed that tumorous growth in animals and birds resulted from "minute" infectious particles. Experiments then supported the hypothesis, that the etiologic agent in many animal tumors was viral. The development of molecular biology, supported by technical advances and conceptual understanding of macromolecular action, led to an explosive increase in studies of animal oncogenic viruses. For a decade, new findings emerged from research laboratories revealing the enormous variety of such agents, the complex- ity of their interactions with cells, and the tantalizingly possible mechanisms by which they might cause malignant transformation of the cell. Repeatedly, clues emerged which suggested the intervention of viral agents in human tumors. A breathless excitement pervaded both the scientific and public communities as highly publicized findings rapidly followed one another. The excitement was no less scientific
出版日期Book 1975
關(guān)鍵詞DNA; carcinogenesis; cell; etiology; leukemia; molecular biology; oncology; tumor; tumorigenesis
版次1
doihttps://doi.org/10.1007/978-1-4684-2733-2
isbn_softcover978-1-4684-2735-6
isbn_ebook978-1-4684-2733-2
copyrightSpringer Science+Business Media New York 1975
The information of publication is updating

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cause malignant transformation of the cell. Repeatedly, clues emerged which suggested the intervention of viral agents in human tumors. A breathless excitement pervaded both the scientific and public communities as highly publicized findings rapidly followed one another. The excitement was no less scientific 978-1-4684-2735-6978-1-4684-2733-2
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Replication and Transformation by Papovavirusesic cycle of replication or a latent infection. For the papilloma viruses it is difficult to obtain a suitable cell line in which the lytic cycle can be studied; therefore, studies that we will discuss concerning viral replication will deal exclusively with SV40 and polyoma.
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ve is our appreciation of the cause of tumors based on observation. Even in the earliest era of the study of infectious diseases, it was proposed that tumorous growth in animals and birds resulted from "minute" infectious particles. Experiments then supported the hypothesis, that the etiologic agent
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New Issues in Financial and Credit Markets” which successfully protected him against a later inoculation with smallpox virus. Although the experiment of Peyrilhe was itself not successful, the idea was kept alive in the minds of a few investigators until the present century, and now viruses as etiological agents in cancer are known to be commonplace.
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