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Titlebook: Cancer Genes; Functional Aspects Enrico Mihich,David Housman Book 1996 Springer Science+Business Media New York 1996 apoptosis.cancer.cell.

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發(fā)表于 2025-3-21 16:32:29 | 只看該作者 |倒序瀏覽 |閱讀模式
書目名稱Cancer Genes
副標題Functional Aspects
編輯Enrico Mihich,David Housman
視頻videohttp://file.papertrans.cn/222/221110/221110.mp4
叢書名稱Pezcoller Foundation Symposia
圖書封面Titlebook: Cancer Genes; Functional Aspects Enrico Mihich,David Housman Book 1996 Springer Science+Business Media New York 1996 apoptosis.cancer.cell.
出版日期Book 1996
關鍵詞apoptosis; cancer; cell; genes; genetics; leukemia; molecular genetics; oncogene; prevention; protein; protein
版次1
doihttps://doi.org/10.1007/978-1-4615-5895-8
isbn_softcover978-1-4613-7704-7
isbn_ebook978-1-4615-5895-8
copyrightSpringer Science+Business Media New York 1996
The information of publication is updating

書目名稱Cancer Genes影響因子(影響力)




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書目名稱Cancer Genes網絡公開度




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書目名稱Cancer Genes讀者反饋學科排名




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Isaac John,Santhakumar Mohan,Philippe Wengerrs (e.g., growth factors) and their receptors are often encoded by a class of genes called proto-oncogenes. Cells also contain several inhibitory proteins, encoded by tumor suppressor genes which act as either censors or pacers (Marx, 1994).
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地板
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p16 Family Inhibitors of Cyclin-Dependent Kinases,ng at a reduced rate. Therefore, the molecular pathways controlling the cell division cycle must inevitably interact with pathways which regulate cell growth, and are a very likely target of oncogenic events. It was not until very recently, however, that experimental evidence became available to bring such a connection to light.
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Retinoblastoma Protein, Gene Expression, and Cell Cycle Control,rs (e.g., growth factors) and their receptors are often encoded by a class of genes called proto-oncogenes. Cells also contain several inhibitory proteins, encoded by tumor suppressor genes which act as either censors or pacers (Marx, 1994).
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J. Drewniak,P. Garlicka,J. Kope?,S. Zawi?lakated by two observations: firstly, in the many cases tested, mutation of the tail such that it can no longer be phosphorylated is sufficient to convert proto-oncogene to oncogene; secondly, mice lacking Csk have constitutively active Src family kinases and die during embryogenesis (reviewed in .).
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https://doi.org/10.1007/978-3-031-29815-8eurological disorder characterized by a slow and progressive demyelination of the peripheral nerves and causing paraparesis and spasticity of the lower extremities. Moreover, HTLV-I can also cause a severe granulocytosis and eosinophilia. and in some cases an autoimmune chronic athritis..
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