Titlebook: Calcium, Neuronal Function and Transmitter Release; Proceedings of the S Rami Rahamimoff,Bernard Katz Conference proceedings 1986 Martinus

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書目名稱Calcium, Neuronal Function and Transmitter Release影響因子(影響力)




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Directed

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Modulation Of Calcium Channels in Cardiac Cells by Neurotransmitters channels. Adrenergic neurotransmitter effects on Ca currents, I. in cardiac cells have served as prime examples of modulation of voltagegated ion channels. Recently it has become possible to analyze molecular events Involved in Ca channel modulation. Ca channels are activated by changes in membrane

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Persistent Inactivation of Potassium Currents by Elevation of Intracellular Calciumtoreceptor, remain reduced for days after associatively conditioning the nudibranch mollusc .. Because intracellular calcium, Ca.. (as measured by differential absorption spectrophotometry of calcium complexed with the dye arsenazo III), undergoes prolonged elevation during the acquisition of the co

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Some Consequences of Intracellular Calcium Binding on Phasic Synaptic Transmitter Releasear junction. Katz & Miledi (1) showed that external calcium was necessary at the time of the first nerve impulse and suggested that calcium entered the terminal and, by binding to some receptor site at the presynaptic terminal, released transmitter. The “residual calcium” hypothesis for facilitation

HARP

Calcium Cooperativity in Calcium Entry and Calcium Action, and its Implications with Regard to Facilrnal medium via voltage-regulated cation-selective channels (reviews, 1, 2, 3). At the vertebrate neuromuscular junction this dependence is manifest in essentially the same way for release provoked by nerve impulses (i.e., end-plate potentials, EPPs) or release manifest as a raised frequency of mini