Titlebook: Calcium Antagonists; Mechanism of Action Nicholas Sperelakis,James B. Caulfield Book 1984 Martinus Nijhoff Publishing, Boston 1984 calcium

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Peristalsis

DEAF

Action of Calcium Slow Channel Inhibitors on Cardiac and Vascular Smooth Muscle MembranesCa.-selective slow channels during the phase 2 of the action potential when these channels are presumably “open.” A number of organic compounds classified as calcium antagonists (1) were found to antagonize the extracellular Ca.-dependent contraction. These compounds are also referred to as “calcium

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Antianginal Effects of Calcium Antagoniststo a patient provides the basis for their increasing use in patients disabled by angina pectoris due to either occlusive coronary artery disease or coronary artery spasm. As discussed elsewhere in this symposium, the relative selectivity of calcium blockers for vascular smooth muscle over cardiac mu

閃光東本

Clinical Electrophysiology of the Calcium Antagonistsvailable until Fleckenstein and colleagues (2) proposed the concept of calcium antagonism. A variety of substances were shown to be capable of inhibiting the transmembrane passage of calcium ions. Although this work had concentrated on the role of calcium in the myocardium the profound influence of

閃光東本

Basic Features of The Frequency- and Voltage-Dependent Block By D600 of Calcium Channels in Ventricuoupling, sinoatrial rhythm, atrioventricular transmission, and certain patterns of dysrhythmia. A variety of organic agents, of which verapamil and D600 (methoxyverapamil) have been best studied, block Ca channels and thereby reduce I. in cardiac tissue (1–4). The consequences of this action include

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