Titlebook: C-Myc in B-Cell Neoplasia; 14th Workshop on Mec Michael Potter (Chief),Fritz Melchers (Director) Book 1997 The Editor(s) (if applicable) an

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B-lymphocyte-lineage Cells from Early Precursors to Ig-secreting Plasma Cells: Targets of Regulation miz-1 (see papers in this volume, in particular Austen et al., Schneider et al., Schreiber-Agus et al.), is thought to control expression of genes involved in the proliferation and differentiation of cells. Very few lineages of cellular development have been studied . and . in comparable detail as

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DNA Conformation, Topology, and the Regulation of , Expressionultitude of physiological processes and has been reported to be regulated by a long list of hormones, cytokines, lymphokines, nutritional status, development and differentiation. Myc levels can be pharmacologically perturbed, at least transiently, by a host of agents. Consistent with the notion that

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Structure and Expression of the c-Myc/Pvt 1 Megagene Locusing Burkitt’s Lymphoma (BL), AIDs-NHL, mouse plasmacytoma (Pct) and possibly multiple myeloma (MM). There is a restriction associated with this Tx such that only the immunoglobulin (Ig) heavy chain gene is found juxtaposed to c-Myc and only the Ig light chain gene is found juxtaposed to Pvt 1. Over

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c-Myc Promotes Survival of WEHI 231 B Lymphoma Cells from Apoptosisssion or inappropriate time of expression of the c-. gene has been found to promote apoptosis. Cleveland and coworkers observed that addition of a vector expressing c-Myc protein accelerated apoptosis following IL-3 deprivation of the 32D Independent myeloid cell line [3]. Similarly Evan and coworke

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Role of c-myc and p27 in Anti-IgM Induced B-Lymphoma Apoptosiso undetectable levels by 8–24 hours. These cells die soon thereafter via apoptosis. IgD receptor crosslinking also leads to an increase in c-myc expression, but it remains above baseline levels for more than 24 hours; these cells continue to proliferate and do not die. We previously reported that an