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Titlebook: Experimental Biology; William L. McGuire Book 1978 Springer Science+Business Media New York 1978 breast cancer.cancer.cell.chemotherapy.ho

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發(fā)表于 2025-3-21 16:33:42 | 只看該作者 |倒序?yàn)g覽 |閱讀模式
期刊全稱Experimental Biology
影響因子2023William L. McGuire
視頻videohttp://file.papertrans.cn/191/190589/190589.mp4
圖書封面Titlebook: Experimental Biology;  William L. McGuire Book 1978 Springer Science+Business Media New York 1978 breast cancer.cancer.cell.chemotherapy.ho
影響因子Breast cancer continues to be the focus of intense basic and clinical research. In Valurne 1 of this series we dealt exclusively with topics concerned with therapy. In the present Valurne 2, we turn our attention to the experimental biology which is the foundation for our understand- ing of problems concerned with breast cancer etiology, mechanisms of hormone action, cell kinetics, experimental chemotherapy, and markers of tumor burden. The contributors to the volume are all noted scholars who are personally investigating these problems. The first chapter addresses the question, do hormones cause breast cancer? Segaloff provides us with a rational up-to-date overview of the existing data. He concludes that hormones by themselves are not tumor initiators but rather alter the hast environment so that other carcinogens are effective. lt is pointed out that the selection of the modeltest system is critical; one can almost assure any desired result by choosing an ap- propriately biased test system. The question of the role of viruses in the etiology of human breast cancer remains unanswered despite elegant studies in mause systems.
Pindex Book 1978
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Preneoplasia in Breast Cancer,rigenesis was proposed and developed by DeOme and co-workers..) The concept states that mammary tumors arise from morphologically discrete epithelial lesions that are altered from normal mammary epithelial cells. This concept of progressive stages in the development of mammary neoplasia was recogniz
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The Cell Kinetics of Mammary Cancers,s chapter will review the available literature of unperturbed breast cancer cell kinetics in both transplantable and spontaneous animal models, as well as the available material on human breast cancers. The data reported here originated from . and short-term . studies performed directly with the tum
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Therapy in Experimental Breast Cancer Models, the tissue or organ of their origin has been paralleled by an increasing interest in experimental animal model systems that not only are representative of human malignancies histologically, but also originate in the organ or tissues, and have the growth and metastasizing characteristics of the part
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