Titlebook: Brain Somatic Cross-Talk and the Central Control of Metabolism; Claude Kordon,Iain Robinson,Yves Christen Conference proceedings 2003 Spri

LAPSE

Imitation Learning with?Sinkhorn Distancesrodents. For some time now, we have been investigating the central site and mechanism of action of GHS, mostly in relation to their growth hormone-releasing action. However, it has become clear that these compounds can activate neuropeptide Y-containing neurones and interact with the hypothalamic ci

不確定

啞巴

Georgios Papagiannis,Yunpeng Lialterations in metabolism resulting in increases in deposition of visceral fat at the expense of muscle. My laboratory is investigating the underlying mechanism of this declining activity of the central nervous system (CNS) and related metabolic alterations. Our objective is to reverse, delay or pre

可互換

Georgios Papagiannis,Yunpeng Liveral galanin receptor subtypes. In the brain, the distribution of GALP-containing cell bodies is restricted to the arcuate nucleus (Arc), median eminence (ME) and infundibular stalk (IS) of the medial basal hypothalamus. In the Arc, GALP neurons express the signaling form of the leptin receptor (Ob

焦慮

ANA

Wendelin B?hmer,Klaus Obermayerprocesses, including immunity and inflammation. Numerous data have accumulated showing a strong interplay between factors of inflammation (cytokines, adipsin for example) and adipose cells. First, preadipocytes or adipocytes of both peripheral or bone marrow origins are able to synthesise and secret

incisive

tattle

細胞膜

Jingkuan Song,Tao He,Hangbo Fan,Lianli Gao leading to hyperinsulinemia, hyperglycemia, and enlarged fatty liver. Our laboratory have developed a mouse model that mimics these features of generalized lipodystrophy: the syndrome occurs in transgenic mice expressing a truncated version of a nuclear protein known as nSREBP-1c (sterol-regulatory

跑過

Adrian Spurr,Emre Aksan,Otmar Hilliges humoral link between metabolic function and the regulation of hypothalamo-pituitary secretions. Absence of leptin production as seen in . mice or in some rare cases of human obesity not only leads to hyperphagia, reduced energy expenditure, and hypercorticism, but also, most importantly, to sterili