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Titlebook: Brain Protection; Morphological, Patho Klaus Wiedemann,Siegfried Hoyer Book 1983 Springer-Verlag Berlin Heidelberg 1983 Arzneitherapie.Barb

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發(fā)表于 2025-3-21 17:44:29 | 只看該作者 |倒序瀏覽 |閱讀模式
期刊全稱Brain Protection
期刊簡稱Morphological, Patho
影響因子2023Klaus Wiedemann,Siegfried Hoyer
視頻videohttp://file.papertrans.cn/191/190227/190227.mp4
圖書封面Titlebook: Brain Protection; Morphological, Patho Klaus Wiedemann,Siegfried Hoyer Book 1983 Springer-Verlag Berlin Heidelberg 1983 Arzneitherapie.Barb
影響因子Significant progress has doubtlessly been made in the field of cere- bral protection compared to earlier centuries, as recently reviewed by Elisabeth Frost (6). She cites the recommendations for treat- ment of brain trauma by Areteus, a Greek physician of the second century A. D. He expressed quite modem views with regard to the need for prompt action considering complications that follow even minor symptoms. He advised burr holes for evacuation of hema- toma in seizures, the use of diuretics and, most interestingly, also hypothermia. German surgeons of the 17th century had little more to offer than prescriptions of which the most effective constituent was alcohol (10). Thus, Sir Astley Cooper was probably the next surgeon to make noteworthy contributions when advising the use of leeches to the temporal artery and other means of bleeding in- stead of surgical intervention in cases of raised intracranial pressure (loc. cit. 6). Although our knowledge has greatly expanded during the last two decades, extensive discussions have led to only few conclusions. Promising results from animal studies were translated to clinical sit- uations only to yield controversial and sometimes confusing
Pindex Book 1983
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Survival of Cortical Neurons After Ischemia: Dependency on Severity and Duration,cy decreases and the amplitude of the EEG becomes isoelectric at about 0.15 ml/g/min (12,15). Somatosensory evoked potentials are nearly unchanged until flow drops below 0.2 ml/g/min. The amplitudes then become smaller and smaller until at flow values of about 0.15 ml/g/min, the evoked potentials ar
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Recovery from Disturbed Cerebral Ion Homeostasis Following Severe Incomplete Ischemia and Modificatral blood flow (GBF) and metabolism has become increasingly accepted. Results from animal studies with different approaches to cerebral ischemic lesion (11, 23, 24) as well as from patients with severe head injuries (9,27) have indicated that pharmacologically induced “l(fā)ow flow and low metabolism” d
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Comparative Evaluation of Barbiturate and CA++Antagonist Attenuation of Brain Free Fatty Acid Libermagnitude unlike that of any other cerebral metabolite known thus far (4,5). Therefore, we hypothesized that brain FFA accumulation may reflect the evolution of ischemic brain injury at least during complete global ischemia. If so, drugs effective in improving the tolerance of the brain to ischemic
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Pathophysiology and Pathobiochemistry of Acute Brain Infarction in the Gerbil: The Influence of Metlinical situation as closely as possible. Among the various models used, the gerbil (Meriones unguiculatus) is of particular interest because in this species the circle of Willis is incomplete, and extracranial carotid artery occlusion results in stroke in a certain percentage of animals (11). A cer
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發(fā)表于 2025-3-23 08:04:51 | 只看該作者
Clinical Trials of Brain Protection: Problems and Solutions, factors — the rapidity with which cerebral insults produce dysfunction, the difficulty in determining how much structural brain damage has occurred, and the irreversibility of changes in neuronal structure once they have occurred. Introduced at first in the form of barbiturate therapy as a protecti
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