Titlebook: Brain Ischemia; Basic Concepts and C Louis R. Caplan (Professor and Chairman) Book 1995 Springer-Verlag London Limited 1995 PET.biochemistr

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Mechanisms of Cell Death in Ischemiatid atherosclerosis. This intervention involves the unilateral interruption of circulation (for up to 30 minutes) through three major vessels: the common, internal and external carotid arteries (Sundt et al. 1981; Morawetz et al. 1984). The resulting circulatory alterations are reflected in ipsilate

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Positron Emission Tomography (PET) in Acute Ischemic Stroke: Pathophysiological and Clinical Implica artery (e.g. the trunk of the middle cerebral artery, MCA) triggers complex regulatory mechanisms that tend to compensate for the decrease in perfusion pressure, such as distal bed vasodilatation and subsequent ‘opening’ of pial anastomoses. This results in a gradient of reduction in cerebral blood

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Excitotoxicity and Strokeiately focused on insult limitation, for example, utilizing thrombolytic therapy in the minutes to hours after the onset of symptoms to restore blood flow in a clogged cerebral artery. However, a complementary approach is to reduce the intrinsic vulnerability of brain parenchyma to hypoxic-ischemic

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Growth Factors in Strokees, including processes of cell survival, growth and division. An increasing number of such factors have been identified during the last decade, most falling into several known gene ‘superfamilies.’ including the neurotrophin, fibroblast growth factor (FGF), transforming growth factor a and (3 (TGF-

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Prefatory Commentse vascular disease. Occasionally, both eyes are involved together or sequentially, causing temporary or even permanent blindness. I include a chapter on eye ischemia in this txt because the anatomy and physiology of the retina and optic nerve are quite distinctive from those of the brain; the anatom

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Ocular Ischemiand to be affected in different ways by different diseases. More importantly, different visual signs and symptoms occur with the involvement of either vascular territory. In general, the retinal vessels are more often affected by embolic disease, which may lead to transient monocular blindness or ret

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Prefatory Commentsg. Physicians during the ensuing years recognized that bleeding due to trauma or reduced blood coagulability could lead to brain hemorrhage. The cause of brain softening, then customarily called ’encephalomalacia’ or ’ramollissements’ was, however, not evident until the time of Virchow, who was prob