Titlebook: Brain Iron Metabolism and CNS Diseases; Yan-Zhong Chang Book 2019 Springer Nature Singapore Pte Ltd. 2019 Iron.Iron metabolism.Brain.Neuro

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書目名稱Brain Iron Metabolism and CNS Diseases影響因子(影響力)




書目名稱Brain Iron Metabolism and CNS Diseases影響因子(影響力)學(xué)科排名




書目名稱Brain Iron Metabolism and CNS Diseases網(wǎng)絡(luò)公開度




書目名稱Brain Iron Metabolism and CNS Diseases網(wǎng)絡(luò)公開度學(xué)科排名




書目名稱Brain Iron Metabolism and CNS Diseases被引頻次




書目名稱Brain Iron Metabolism and CNS Diseases被引頻次學(xué)科排名




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書目名稱Brain Iron Metabolism and CNS Diseases年度引用學(xué)科排名




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書目名稱Brain Iron Metabolism and CNS Diseases讀者反饋學(xué)科排名

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978-981-13-9591-8Springer Nature Singapore Pte Ltd. 2019

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,Spezielle Matrix- und L?sungsfunktionen,t of oxygen. As a component of various enzymes, it participates in the tricarboxylic acid cycle and oxidative phosphorylation. Iron in the nervous system is also involved in the metabolism of catecholamine neurotransmitters and is involved in the formation of myelin. Therefore, iron metabolism needs

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Analytic and Algebraic Families, in the brain. Brain iron metabolism disorders might be one cause of neurodegenerative diseases. This review mainly summarizes the normal process of iron entry into the brain across the blood–brain barrier, and the distribution and transportation of iron among neurons and glial cells, as well as the

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Uniqueness of meromorphic functions,earance of deposit of neurofibrillary tangles. Increasing evidence suggests that disorders of metal ion metabolism in the brain are one of the risk factors for the pathogenesis of AD. Iron, one of the endogenous metal ions, involves in many important physiological activities in the brain. Iron metab

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Aldo Altamore,Francesco Poppi,Sabino Maffeo, mainly, heart, and endocrine pancreas. It is an autosomal recessive disease caused by a GAA triplet-repeat localized within an Alu sequence element in intron 1 of frataxin (.) gene, which encodes a mitochondrial protein FXN. This protein is essential for mitochondrial function by the involvement o

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Mes premieres constructions de programmes, brainstem, and spinal cord. The etiology and pathogenesis of this devastating disease remain largely unknown. Increasing evidence suggests that iron accumulation is involved in the onset and progression of ALS. In this review, we discuss the regulation of iron homoeostasis in the brain, the misreg