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Titlebook: Brain Edema IX; Proceedings of the N Umeo Ito (Director and Neurosurgeon in Chief),Alex Conference proceedings 1994 Springer-Verlag/Wien 19

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發(fā)表于 2025-3-30 11:49:24 | 只看該作者
https://doi.org/10.1007/0-387-31804-6nses to the disease. We have developed a safe, easily used rat model for . type b meningitis. We measured cerebral blood flow, glucose utilisation and second messenger activity in this model, and carried out parallel light and ultrastructural analysis of glial and neuronal responses. Only protein ki
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發(fā)表于 2025-3-30 12:50:40 | 只看該作者
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發(fā)表于 2025-3-30 23:23:10 | 只看該作者
Valeria Panebianco,Jurgen J. Fütterer minutes of 2 mM glutamate, with or without glutamate antagonists (dizocilpine, kynurenate, or NBQX), were allowed to recover for 60 minutes. The protocol was identical to that in noninvasive multinuclear NMR spectroscopy studies (.P/.H/.F) of live slices. Percent water and swelling were determined
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發(fā)表于 2025-3-31 03:12:53 | 只看該作者
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978-3-7091-9336-5Springer-Verlag/Wien 1994
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發(fā)表于 2025-3-31 19:22:19 | 只看該作者
Acta Neurochirurgica Supplementhttp://image.papertrans.cn/b/image/190161.jpg
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發(fā)表于 2025-4-1 00:56:47 | 只看該作者
Neuron-Glial Interaction During Injury and Edema of the CNSators are glutamate, acidosis, free fatty acids, or high extracellular potassium. Glial homeostatic mechanisms are activated to prevent the secondary injury from these mediators. The glial clearance mechanisms have been studied in detail using in vitro systems allowing for a close control of the gli
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