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Titlebook: Bradykinin, Kallidin and Kallikrein; Supplement Ervin G. Erd?s Book 1979 Springer-Verlag Berlin Heidelberg 1979 Bradykinin.Histamin.Kallidi

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發(fā)表于 2025-3-21 18:07:12 | 只看該作者 |倒序瀏覽 |閱讀模式
期刊全稱Bradykinin, Kallidin and Kallikrein
期刊簡稱Supplement
影響因子2023Ervin G. Erd?s
視頻videohttp://file.papertrans.cn/191/190118/190118.mp4
學科分類Handbook of Experimental Pharmacology
圖書封面Titlebook: Bradykinin, Kallidin and Kallikrein; Supplement Ervin G. Erd?s Book 1979 Springer-Verlag Berlin Heidelberg 1979 Bradykinin.Histamin.Kallidi
影響因子Volume XXV of the Handbook of Experimental Pharmacology series entitled "Bradykinin, Kallidin, and Kallikrein" was published in 1970. My aim in editing this volume of the series is not to replace, but to update the 1970 edition. During the decade preceding the publication of Vol. XXV, the existence of kinins and kallikreins gained acceptance, the protein components of the system were purified and characterized and the peptides were synthesized. Even after these accomplish- ments, interest in the subject has not abated, but has increased substantially. We have learned a great deal about the role that components of the kallikrein-kinin system play in other systems and about the immensely complex and intricate inter- actions in blood. Directly or indirectly, kallikrein and kinins affect the coagulation of blood, the activation of complement, and the generation of angiotensin. Kinins release or modulate the actions of other agents, including prostaglandins, histamine, and catecholamines. Inhibitors of kallikrein or kininase II are employed, for example, in extracorporeal circulation or in hypertension. Kallikrein, kinins, and kininases, present in urine, were described first in 1925 an
Pindex Book 1979
The information of publication is updating

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Kininogenases of Blood Cells (Alternate Kinin Generating Systems),of kinin generating systems that are present in the body. The generating systems include a variety of normal and pathological tissues and fluids. Because of the involvement with the coagulation system, the plasma system will generate kinins very rapidly upon injury or disease and still must be consi
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Kallikrein Inhibitors,as often been indicated by yields of more than 100% during the first isolation steps of the enzyme. Thus, the mechanism of inhibition of proteinases in biologic systems is not unique as formerly thought.
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Kinins and the Peripheral and Central Nervous Systems,on their ability to stimulate chemoreceptors for pain in animals and especially in man (., 1970). Direct application of bradykinin to an exposed cantharidin blister area on the forearm of human subjects causes a slightly delayed, burning type of pain. This reaches maximum in about 1 min and graduall
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Release of Vasoactive Substances by Kinins,he release of other vasoactive substances. The potential of kinins to activate the prostaglandin synthesizing system in the kidney (. et al., 1972), pregnant uterus (. et al., 1974), and vasculature (. et al., 1975; . et al., 1975) deserves special attention. This activation of synthesis results in
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