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Titlebook: Bone Regulators and Osteoporosis Therapy; Paula H. Stern Book 2020 Springer Nature Switzerland AG 2020 Bone.Bone cell regulators.Osteoporo

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樓主: malcontented
31#
發(fā)表于 2025-3-27 00:00:32 | 只看該作者
Cytokines and Bone: Osteoimmunology, the diverse effects that cytokines and inflammation have on osteoblasts, osteoclasts, and bone mass. This chapter provides a broad overview of this topic with extensive references so that, if desired, readers can access specific references to delve into individual topics in greater detail.
32#
發(fā)表于 2025-3-27 03:09:40 | 只看該作者
0171-2004 of bone disorders and discusses the pathophysiology of osteo.This volume is designed to provide an understanding of current and potential therapies for osteoporosis. The opening chapter introduces the? cells of bone and their interactions. ?Several following chapters describe factors affecting bone
33#
發(fā)表于 2025-3-27 06:05:21 | 只看該作者
34#
發(fā)表于 2025-3-27 12:49:25 | 只看該作者
Thyroid Hormones, Glucocorticoids, Insulin, and Bone,eral pathways. Their endogenous or exogenous excess will induce pathological processes that might elevate the risk of fractures. Insulin and the carbohydrate metabolism elicit a physiological effect on bone; however, the lack of insulin (type 1 diabetes) or insulin resistance (type 2 diabetes) have deleterious influence on bone tissue.
35#
發(fā)表于 2025-3-27 17:07:23 | 只看該作者
36#
發(fā)表于 2025-3-27 18:03:01 | 只看該作者
37#
發(fā)表于 2025-3-27 23:20:16 | 只看該作者
Vascular Disorders of the?Liverary aspects of how PTH and PTHrP behave and as a call to encourage further investigation that will yield more comprehensive understanding of the physiological and pathological steps at play, with a goal toward novel therapeutic interventions.
38#
發(fā)表于 2025-3-28 02:26:20 | 只看該作者
https://doi.org/10.1007/978-3-030-79132-2chemokines in altering the bone microenvironment through effects on angiogenesis and tumor invasion is discussed. Very few therapeutic agents that influence bone formation by targeting chemokines or chemokine receptors are available, although a few are currently being evaluated.
39#
發(fā)表于 2025-3-28 08:22:18 | 只看該作者
Nonalcoholic Fatty Liver Disease matrix. In contrast, deficient actions of FGF23 result in hyperphosphatemic tumoral calcinosis with high 1,25-dihydroxyvitamin D levels. These results indicate that FGF23 is a physiological regulator of phosphate and vitamin D metabolism and indispensable for the maintenance of serum phosphate levels.
40#
發(fā)表于 2025-3-28 11:33:06 | 只看該作者
PTH and PTHrP Actions on Bone,ary aspects of how PTH and PTHrP behave and as a call to encourage further investigation that will yield more comprehensive understanding of the physiological and pathological steps at play, with a goal toward novel therapeutic interventions.
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