Titlebook: Bone Metastases from Prostate Cancer; Biology, Diagnosis a Francesco Bertoldo,Francesco Boccardo,Riccardo Val Book 2017 Springer Internatio

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Imaging of Glycolysis with 18F-FDG PET,alignancy-induced hypermetabolism is generally based upon overexpression of cellular membrane glucose transporters (mainly glucose transporter 1, GLUT-1) and enhanced hexokinase enzymatic activity in tumors [1, 2].

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Affiliation

Studies in Human Ecology and Adaptationntigraphy or the distribution of BMTs (axial vs appendicular) with survival in patients with advanced prostate cancer (PC) [1, 2]. The number of BMTs has recently been evaluated as a prognostic predictor [3]. Patients with metastatic castration-resistant PC with a higher number of BMTs had a shorter

hypotension

https://doi.org/10.1007/978-3-319-64407-3e formation of new bone by osteoblasts. Local and systemic growth factors regulate the differentiation and activity of the osteoclasts and osteoblasts (and osteocytes). Maintenance and repair of normal bone result in the release of enzymes, peptides and mineral components that have been characterise

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Miscellaneous Tools/Implements,cuss the contemporary notion of tumor markers from a general point of view. We focus on some specific characteristics of prostate-specific antigen (PSA), showing why it is the sole circulating tumor marker presently recommended in the follow-up of patients – with or without metastases – treated with

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Gudgeon

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analogous

,Durchführung der Lithiumtherapie,-18F-fluorocyclobutane-1-carboxylic acid (.F-FACBC) or probe targeting the prostate-specific membrane antigen (PSMA). These radiotracers showed in literature better performance in the detection of prostate cancer recurrence as compared to choline PET/CT imaging [1, 2].