Titlebook: Blood-Brain Barrier; Drug Delivery and Br David Kobiler,Shlomo Lustig,Shlomo Shapira Book 2001 Springer Science+Business Media New York 200

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書目名稱Blood-Brain Barrier影響因子(影響力)




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Book 2001ia, ischemia, trauma, hypertension, inflammation and epilepsy. Recent years research has partially elucidated the mechanisms underlying the development of some of these brain disorders as well as the pathways used by different pathogens, like bacteria and viruses, to initiate eNS infections. The dev

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Novel Endothelial-Mediated Responses Associated with Microcirculation and BBB Function,7). Our initial studies demonstrated that NO was involved in the postischemic hypoperfusion observed in animal models of cerebral ischemia (Spatz, .,1995b). This effect was related to increased levels of ET-1 in the cerebrospinal fluid (CSF). Treatment with ET-1 receptor antagonists abolished the hy

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Modulators of Blood-Brain Barrier (BBB) Permeability: , and , Drug Transport to the Brain,ult in increased or decreased paracellular and/or transcellular transport of compounds to the brain. Under such conditions, upregulated transcellular transport may provide a key to target drugs selectively to the BBB and subsequently into the brain. In the BBB Research Group of the Division of Pharm

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Controlled Modulation of the Blood-Brain and Blood-Tumor Barrier Using Novel Lipid Mimetic Compoundl transiently open the BBB and facilitate entry of a particular drug or agent to the brain (Abbot). Modulation of the BBB with bioactive molecules in contrast to osmotic disruption is appearing as an effective technique. Some limited clinical success has been reported with this approach in the perme

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Stress, Pyridostigmine and the Blood Brain Barrier,ition, fatigue, sleep disturbance, confusion), which could not be easily explained by the effects of a compound that does not enter the CNS. The controversy around the “Gulf war syndrome” was further complicated by the suggestion that stress exposure (physical conditions, combat related anxiety, per

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