Titlebook: Blood Coagulation Disorders; Hemorrhagic Diathese Fritz Reinhard Matthias Book 19871st edition Springer-Verlag Berlin Heidelberg 1987 blood

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Physiology of Blood Coagulation and Hemostasissel wall. After local injury of the vascular system the extravasation of blood is stopped by the formation of a hemostatic plug, and after thrombosis a dissolution with total or partial reconstitution of the affected vascular section is attained through the fibrinolytic system. In these processes, v

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The Diagnosis of Coagulation Disordersium from normal coagulability which exceeds a certain level. Depending on the extent of the disorder in the coagulation and fibrinolysis system and the availability of inhibitors of coagulation and fibrinolysis, the hypocoagulability or hypercoagulability may take a clinically latent course or becom

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Thrombocytic Hemorrhagic Diathesesn disorder). There may be reduced formation (thrombocytopenia), increased production (thrombocytosis or thrombocythemia), or functional defects (thrombocytopathy). They can also be congenital or acquired. There can also be increased platelet turnover (turnover disorder), which can be the cause of a

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Vascular Hemorrhagic Diatheses (Table 3). The bleeding types correspond to those in thrombocytopenia and thrombocytopathies; the bleeding is mainly petechial. Bleeding after trivial injuries, nasal bleeding, bleeding after tooth extraction or operation, and menorrhagia are typical characteristics. The localized vascular malforma

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Coronary Thrombosis and Myocardial Infarctionith or without a disseminated destruction of the cardiac musculature or a myocardial infarction with a localized loss of myocardial function. One of the most important preconditions for the occurrence of an acute cardiac infarction is a stenosis of a coronary artery of 75% or more of its cross-secti

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