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Titlebook: Blistering Diseases; Clinical Features, P Dédée F. Murrell Book 2015 Springer-Verlag Berlin Heidelberg 2015 Epidermolysis bullosa.Immunoflu

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發(fā)表于 2025-3-21 19:26:51 | 只看該作者 |倒序?yàn)g覽 |閱讀模式
期刊全稱(chēng)Blistering Diseases
期刊簡(jiǎn)稱(chēng)Clinical Features, P
影響因子2023Dédée F. Murrell
視頻videohttp://file.papertrans.cn/190/189156/189156.mp4
發(fā)行地址Describes all the dermatologist needs to know about blistering diseases.Presents information in a readily intelligible and accessible way.Includes algorithms for treatment and for testing and monitori
圖書(shū)封面Titlebook: Blistering Diseases; Clinical Features, P Dédée F. Murrell Book 2015 Springer-Verlag Berlin Heidelberg 2015 Epidermolysis bullosa.Immunoflu
影響因子This book is a comprehensive compendium of current knowledge on inherited and autoimmune blistering diseases that relates advances in our understanding of the pathogenetic mechanisms to management of the individual diseases. The aim is to provide a detailed reference for dermatologists who care for patients with these conditions and a useful “one-stop information shop” for specialists outside of dermatology. The book opens by describing the structure and biology of the epidermis and basement membrane zone and discussing the genes and proteins that are targets for mutations and autoantibodies. The role of the various diagnostic tests is explained, and clinical manifestations of the specific diseases are presented with the aid of many high-quality illustrations. The forms of treatment appropriate in specific conditions are then described in depth, with coverage of dressings, drugs, surgical procedures, gene therapy, and other novel approaches. Helpful algorithms are included both for testing and monitoring and for treatment.
Pindex Book 2015
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書(shū)目名稱(chēng)Blistering Diseases影響因子(影響力)




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The Immune System in Normal Skin and in AIBDnment, it protects against hazardous substances, such as chemicals and physical stress. Various immune cells reside in the two main compartments of the skin, the epidermis and the dermis, under physiological steady-state conditions. It is essential for maintaining the integrity of the skin that immu
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Desmosomal Proteins as Autoantigens in Pemphigusere are several variants of pemphigus, each with unique clinical, histological, and immunologic features. Interestingly, when different desmosomal proteins are targeted by the autoimmune response, different clinical and histological features are seen. In this chapter we review the key intercellular
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Collagen XVII and Its Role in Junctional Epidermolysis Bullosathelial cells to the underlying basement membrane. It is a homotrimeric type II transmembrane protein consisting of three 180 kDa alpha-1 (XVII) chains. Each individual chain is encoded by the . gene. Mutations in . usually lead to loss of collagen XVII and a phenotype comprising congenital generali
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Kindlin-1 and Its Role in Kindler Syndromedhesions, which serve as supramolecular signalling and actin-anchoring platforms. Together with talin, kindlins directly bind to integrin β subunit cytoplasmic tails and are responsible for integrin activation. Mutations in the kindlin-1 gene cause the Kindler syndrome, a rare genodermatosis charact
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