Titlebook: Biopsy pathology of muscle; Michael Swash,Martin S. Schwartz Book 1984 Springer Science+Business Media New York 1984 biopsy.electron micro

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Muscular dystrophies,nges in skeletal muscle fibres. Most begin in childhood but in others the disease is not recognized until adult life. Classification depends on clinical, genetic and histological criteria (Table 6.1). The childhood myopathies are, by convention, classified separately since these disorders are only v

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,‘Benign’ myopathies of childhood,sometimes referred to as ., although in some instances they may not cause symptoms until later in childhood or even until adult life. These disorders usually have a genetic basis, although this may not be apparent. In most patients weakness is mild or moderate, and marked wasting is unusual. These d

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Metabolic, endocrine and drug-induced myopathies,hare a common pathogenetic mechanism. The metabolic myopathies result from a biochemical defect in muscle metabolism itself, the endocrine myopathies from an abnormal hormonal environment, probably affecting muscle metabolism, and the drug-induced myopathies from a direct toxic effect of the drug on

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Neurogenic disorders,eural pathways, other than the lower motor neuron. For example, in motor neuron disease there is almost always involvement of the upper motor neuron and in peripheral neuropathies sensory fibres are also involved. In this chapter only the changes seen in muscle biopsies will be described. A general

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Interpretation of the muscle biopsy,hed without them by using formalin-fixed, paraffin-embedded material stained with HE alone or with additional slides prepared by the PAS, PTAH or van Giesen techniques. For example, typical cases of untreated polymyositis in which inflammation is prominent pose no difficulty in diagnosis, and there

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