Titlebook: Biophysics of the Pancreatic β-Cell; Illani Atwater,Eduardo Rojas,Bernat Soria Book 1986 The Editor(s) (if applicable) and The Author(s),

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spinal-stenosis

GEST

Guatemala, an Alternation in Continuitysmitters. Examples include secretory granules from chromaffin cells,. pancreatic B-cells,. platelets. and mast cells. We have recently shown. that it is possible to obtain real-time measurements of the kinetics of secretion of vesicle contents by monitoring ATP release from stimulated medullary chro

Malleable

Cultivate

https://doi.org/10.1007/978-94-007-0504-3e recent review by Henquin & Meissner.). Microelectrode recordings have shown that in the absence of glucose the β-cell is electrically silent. Increasing plasma glucose depolarises the membrane by an amount that is dependent on the glucose concentration and electrical activity is initiated when thi

預(yù)測(cè)

,Adult ESL Immigrant Learners …, the cell, mobilizes calcium from stores within the cell and thus triggers the exocytosis of insulin.. Calcium uptake occurs when glucose depolarizes the β-cell membrane to trigger periodic bursts of calcium action potentials.. The mechanisms which couple glucose metabolism to the depolarization hav

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https://doi.org/10.1007/978-3-031-46105-7 is to evoke membrane depolarization. followed by a cyclical pattern of electrical activity consisting of slow waves of depolarization with action potential-like spikes.. Tracer flux studies have indicated that glucose metabolism is associated with a decrease in membrane K. permeability. and Ashcrof

albuminuria

Stable-Angina

https://doi.org/10.1007/978-3-031-56118-4al models of diabetes mellitus.. They all have hyperglycemia, hyperinsulinemia, and obesity as common metabolic characteristics.. Among these mouse mutants, particular attention has been paid to the homozygous diabetes mouse carrying the diabetes gene (commonly known as the db/db mouse), and the hom