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Titlebook: Biomolecular Interfaces; Interactions, Functi Ariel Fernández Stigliano Textbook 2015 Springer International Publishing Switzerland 2015 Bi

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發(fā)表于 2025-3-23 13:10:19 | 只看該作者
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發(fā)表于 2025-3-23 15:21:42 | 只看該作者
Wrapping Drug Combinations for Therapeutic Editing of Side Effects: Systems Biology Meets Wrapping om the highly diverse cellular contexts wherein a protein may constitute a desirable or undesirable target. While dehydron wrapping enables the control of specificity, it may not be able to exclude every single toxicity-related target, especially if the latter shares with the therapeutically relevan
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發(fā)表于 2025-3-23 18:47:08 | 只看該作者
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發(fā)表于 2025-3-24 03:29:52 | 只看該作者
High-Level Quantum Chemistry Empowers the Wrapping Technology for Drug Design,her empower the paradigmatic concept of “dehydron-wrapping drug.” This type of analysis provides the required guidance for the incorporation of halogens as wrapping groups in the drug?chemical scaffold. The chapter explores the possibility that the group that wraps exogenously a dehydron upon drug b
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發(fā)表于 2025-3-24 09:06:50 | 只看該作者
Textbook 2015peutic drugs and to allow substantive advances in targeted molecular medicine. This book will be of interest to scientists, students and practitioners in the fields of chemistry, biophysics and biomedical engineering..
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發(fā)表于 2025-3-24 13:40:39 | 只看該作者
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發(fā)表于 2025-3-24 16:40:13 | 只看該作者
Textbook 2015ully understood at the molecular level without considering interfacial behavior..The author presents conceptual advances in molecular biophysics that herald the advent of a new discipline, .epistructural biology., centered on the interactions of water and bio molecular structures across the interfac
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發(fā)表于 2025-3-24 19:06:14 | 只看該作者
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發(fā)表于 2025-3-25 00:47:52 | 只看該作者
Dennis S. Bernstein,David C. Hylandse and natural killer cell cytotoxicity compromised by the original drug treatment. The redesign strategy enables us to create synergies that may empower drug-based anticancer therapy, aligning the immune response with the molecularly targeted treatment?while retaining the anticancer efficacy of the parental compound.
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