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Titlebook: Biomarkers in Neuropsychiatry; A Primer Antonio L. Teixeira,Natalia P. Rocha,Michael Berk Book 2023 The Editor(s) (if applicable) and The A

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發(fā)表于 2025-3-28 16:31:12 | 只看該作者
42#
發(fā)表于 2025-3-28 22:40:25 | 只看該作者
Genetic Biomarkers of Psychiatric Disorders,r decades of linkage and candidate gene studies, technological advancements have allowed for the recent discovery of many common and rare genomic . significantly involved with the heritability of schizophrenia, bipolar disorder, autism spectrum disorder, major depressive disorder, suicide, and many
43#
發(fā)表于 2025-3-29 01:24:01 | 只看該作者
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發(fā)表于 2025-3-29 05:56:07 | 只看該作者
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發(fā)表于 2025-3-29 09:00:46 | 只看該作者
Functional Neuroimaging Biomarkers,ment of biomarkers for neuropsychiatric disorders. It highlights the strengths and limitations of fMRI, reviews current literature on functional neuroimaging biomarkers, explores the challenges in locating clinically relevant biomarkers, and discusses potential applications of functional neuroimagin
46#
發(fā)表于 2025-3-29 14:48:46 | 只看該作者
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發(fā)表于 2025-3-29 16:57:14 | 只看該作者
Digital Markers of Mental Health Problems: Phenotyping Across Biological, Psychological, and Enviroof portable digital devices. Through sensors (e.g., geolocating system) and human-device interactions (e.g., keystrokes), smartphones and wearable devices can be used to generate digital indices that aim to capture a person’s mental health states and mental health determinants across biological, psy
48#
發(fā)表于 2025-3-29 20:27:33 | 只看該作者
49#
發(fā)表于 2025-3-30 00:08:00 | 只看該作者
Biomarkers of Delirium and Cognitive Impairment, and neurotransmitter dysregulation. Currently, the diagnosis of delirium is based on clinical assessment with the support of nonspecific biomarkers. No specific biomarker has been validated or implemented to this date. Nevertheless, many promising biomarkers have been identified in recent studies,
50#
發(fā)表于 2025-3-30 07:16:58 | 只看該作者
,Fluid-Based Biomarkers of Alzheimer’s Disease,iagnosis of AD is of utmost importance because of the irreversible nature of the related neuropathological damage. Decreased levels of amyloid-beta 42 (Aβ.) and elevated levels of total tau and phosphorylated tau (p-tau) in cerebrospinal fluid (CSF) or positive positron-emission tomography (PET) amy
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